PMC:7796052 / 9894-10241 JSONTXT

{"project":"LitCovid-PubTator","denotations":[{"id":"276","span":{"begin":127,"end":132},"obj":"Species"},{"id":"278","span":{"begin":203,"end":213},"obj":"Chemical"},{"id":"281","span":{"begin":148,"end":170},"obj":"Disease"}],"attributes":[{"id":"A276","pred":"tao:has_database_id","subj":"276","obj":"Tax:9606"},{"id":"A278","pred":"tao:has_database_id","subj":"278","obj":"MESH:D020849"},{"id":"A281","pred":"tao:has_database_id","subj":"281","obj":"MESH:D002318"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"To support this hypothesis, in clinical trials aimed at understanding the effect of hormone replacement therapy for menopausal women on the risk of cardiovascular disease, in contrast to other compounds raloxifene did not exhibit a proinflammatory profile [35,36] that could, in part, be explained by its antagonistic effect on the BK B2 receptor."}

{"project":"LitCovid-PD-HP","denotations":[{"id":"T14","span":{"begin":148,"end":170},"obj":"Phenotype"}],"attributes":[{"id":"A14","pred":"hp_id","subj":"T14","obj":"http://purl.obolibrary.org/obo/HP_0001626"}],"text":"To support this hypothesis, in clinical trials aimed at understanding the effect of hormone replacement therapy for menopausal women on the risk of cardiovascular disease, in contrast to other compounds raloxifene did not exhibit a proinflammatory profile [35,36] that could, in part, be explained by its antagonistic effect on the BK B2 receptor."}

{"project":"LitCovid-sentences","denotations":[{"id":"T65","span":{"begin":0,"end":347},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"To support this hypothesis, in clinical trials aimed at understanding the effect of hormone replacement therapy for menopausal women on the risk of cardiovascular disease, in contrast to other compounds raloxifene did not exhibit a proinflammatory profile [35,36] that could, in part, be explained by its antagonistic effect on the BK B2 receptor."}