PMC:7652766 / 21923-22153 JSONTXT

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    LitCovid-PubTator

    {"project":"LitCovid-PubTator","denotations":[{"id":"817","span":{"begin":37,"end":49},"obj":"Disease"},{"id":"841","span":{"begin":100,"end":104},"obj":"Gene"},{"id":"842","span":{"begin":198,"end":201},"obj":"Gene"},{"id":"843","span":{"begin":203,"end":209},"obj":"Gene"},{"id":"844","span":{"begin":216,"end":220},"obj":"Gene"},{"id":"845","span":{"begin":222,"end":229},"obj":"Gene"},{"id":"858","span":{"begin":105,"end":107},"obj":"Gene"},{"id":"859","span":{"begin":84,"end":94},"obj":"Species"}],"attributes":[{"id":"A817","pred":"tao:has_database_id","subj":"817","obj":"MESH:D007249"},{"id":"A841","pred":"tao:has_database_id","subj":"841","obj":"Gene:59272"},{"id":"A842","pred":"tao:has_database_id","subj":"842","obj":"Gene:1636"},{"id":"A843","pred":"tao:has_database_id","subj":"843","obj":"Gene:183"},{"id":"A844","pred":"tao:has_database_id","subj":"844","obj":"Gene:59272"},{"id":"A845","pred":"tao:has_database_id","subj":"845","obj":"Gene:284"},{"id":"A858","pred":"tao:has_database_id","subj":"858","obj":"Gene:6999"},{"id":"A859","pred":"tao:has_database_id","subj":"859","obj":"Tax:2697049"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"s in vasodilation and attenuation of inflammation (85).\nTherefore, considering that SARS-CoV-2 uses ACE2 to enter cells, the main hypothesis of pulmonary pathology is that the increased activity of ACE (Ang-II) over ACE2 (Ang-1-7)"}