PMC:7565665 / 42813-43013
Annnotations
LitCovid-PD-FMA-UBERON
Id | Subject | Object | Predicate | Lexical cue | fma_id |
---|---|---|---|---|---|
T289 | 96-101 | Body_part | denotes | cells | http://purl.org/sig/ont/fma/fma68646 |
LitCovid-PD-MONDO
Id | Subject | Object | Predicate | Lexical cue | mondo_id |
---|---|---|---|---|---|
T350 | 93-95 | Disease | denotes | CF | http://purl.obolibrary.org/obo/MONDO_0009061 |
LitCovid-PD-CLO
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T485 | 96-101 | http://purl.obolibrary.org/obo/GO_0005623 | denotes | cells |
T486 | 120-129 | http://purl.obolibrary.org/obo/CLO_0001658 | denotes | activated |
T487 | 133-134 | http://purl.obolibrary.org/obo/CLO_0001021 | denotes | B |
T488 | 135-144 | http://purl.obolibrary.org/obo/SO_0000418 | denotes | signaling |
LitCovid-PD-CHEBI
Id | Subject | Object | Predicate | Lexical cue | chebi_id |
---|---|---|---|---|---|
T229 | 130-132 | Chemical | denotes | NF | http://purl.obolibrary.org/obo/CHEBI_141424|http://purl.obolibrary.org/obo/CHEBI_25573|http://purl.obolibrary.org/obo/CHEBI_1224 |
LitCovid-PubTator
Id | Subject | Object | Predicate | Lexical cue | tao:has_database_id |
---|---|---|---|---|---|
1070 | 130-134 | Gene | denotes | NFκB | Gene:4790 |
LitCovid-PD-HP
Id | Subject | Object | Predicate | Lexical cue | hp_id |
---|---|---|---|---|---|
T149 | 73-89 | Phenotype | denotes | oxidative stress | http://purl.obolibrary.org/obo/HP_0025464 |
LitCovid-PD-GO-BP
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T578 | 135-144 | http://purl.obolibrary.org/obo/GO_0023052 | denotes | signaling |
T579 | 167-176 | http://purl.obolibrary.org/obo/GO_0016236 | denotes | autophagy |
T580 | 167-176 | http://purl.obolibrary.org/obo/GO_0006914 | denotes | autophagy |
LitCovid-sentences
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T227 | 0-200 | Sentence | denotes | Several reports have suggested that the inherently elevated inflammatory-oxidative stress in CF cells, primarily due to activated NFκB signaling, could be dampened by autophagy augmentation [124,160]. |