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PMC:7565665 / 23745-24016 JSONTXT

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LitCovid-PD-FMA-UBERON

Id Subject Object Predicate Lexical cue fma_id
T138 220-225 Body_part denotes lungs http://purl.org/sig/ont/fma/fma68877
T139 235-243 Body_part denotes neonates http://purl.org/sig/ont/fma/fma292381

LitCovid-PD-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T188 52-55 Disease denotes ALI http://purl.obolibrary.org/obo/MONDO_0006502
T189 85-111 Disease denotes bronchopulmonary dysplasia http://purl.obolibrary.org/obo/MONDO_0019091
T190 113-116 Disease denotes BPD http://purl.obolibrary.org/obo/MONDO_0001156|http://purl.obolibrary.org/obo/MONDO_0019091
T192 261-264 Disease denotes BPD http://purl.obolibrary.org/obo/MONDO_0001156|http://purl.obolibrary.org/obo/MONDO_0019091

LitCovid-PD-CLO

Id Subject Object Predicate Lexical cue
T264 144-145 http://purl.obolibrary.org/obo/CLO_0001020 denotes a
T265 220-225 http://www.ebi.ac.uk/efo/EFO_0000934 denotes lungs
T266 229-234 http://purl.obolibrary.org/obo/NCBITaxon_9606 denotes human

LitCovid-PubTator

Id Subject Object Predicate Lexical cue tao:has_database_id
585 17-23 Species denotes murine Tax:10090
586 229-234 Species denotes human Tax:9606
604 34-43 Disease denotes hyperoxia MESH:D018496
605 85-111 Disease denotes bronchopulmonary dysplasia MESH:D001997

LitCovid-PD-HP

Id Subject Object Predicate Lexical cue hp_id
T79 52-55 Phenotype denotes ALI http://www.orpha.net/ORDO/Orphanet_178320
T80 113-116 Phenotype denotes BPD http://purl.obolibrary.org/obo/HP_0012076
T81 261-264 Phenotype denotes BPD http://purl.obolibrary.org/obo/HP_0012076

LitCovid-PD-GO-BP

Id Subject Object Predicate Lexical cue
T330 119-128 http://purl.obolibrary.org/obo/GO_0016236 denotes autophagy
T331 119-128 http://purl.obolibrary.org/obo/GO_0006914 denotes autophagy
T332 193-202 http://purl.obolibrary.org/obo/GO_0016236 denotes autophagy
T333 193-202 http://purl.obolibrary.org/obo/GO_0006914 denotes autophagy

LitCovid-sentences

Id Subject Object Predicate Lexical cue
T125 0-271 Sentence denotes Additionally, in murine models of hyperoxia-induced ALI, which resembles features of bronchopulmonary dysplasia (BPD), autophagy is proposed as a protective mechanism, and markers of defective autophagy are found in the lungs of human neonates with established BPD [128].