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PMC:7556165 / 58407-58888 JSONTXT

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LitCovid-sample-PD-NCBITaxon

Id Subject Object Predicate Lexical cue ncbi_taxonomy_id
T345 118-128 Species denotes SARS-CoV-2 NCBItxid:2697049
T346 118-122 Species denotes SARS NCBItxid:694009

LitCovid-sample-sentences

Id Subject Object Predicate Lexical cue
T356 0-139 Sentence denotes On the whole, there is still uncertainty about the relationship between ACE2 density on cell membrane and the fate of SARS-CoV-2 infection.
T357 140-481 Sentence denotes An interesting speculation which could reconcile the two position comes from the hypothesis that while ACE2 is for sure the entry door for the virus, once the infection has evolved, there is the subsequent downregulation of ACE2, responsible for the precipitating of respiratory distress, as showed also in animal models (Kuba et al., 2005).

LitCovid-sample-Pubtator

Id Subject Object Predicate Lexical cue pubann:denotes
1611 72-76 Gene denotes ACE2 Gene:59272
1612 243-247 Gene denotes ACE2 Gene:59272
1613 364-368 Gene denotes ACE2 Gene:59272
1614 93-101 Gene denotes membrane Gene:43740571
1615 118-138 Disease denotes SARS-CoV-2 infection MESH:C000657245
1616 299-308 Disease denotes infection MESH:D007239
1617 407-427 Disease denotes respiratory distress MESH:D012128

LitCovid-sample-UniProt

Id Subject Object Predicate Lexical cue uniprot_id
T5766 72-76 Protein denotes ACE2 https://www.uniprot.org/uniprot/Q9UFZ6
T5767 243-247 Protein denotes ACE2 https://www.uniprot.org/uniprot/Q9UFZ6
T5768 364-368 Protein denotes ACE2 https://www.uniprot.org/uniprot/Q9UFZ6

LitCovid-sample-PD-IDO

Id Subject Object Predicate Lexical cue
T222 88-92 http://purl.obolibrary.org/obo/CL_0000000 denotes cell
T223 129-138 http://purl.obolibrary.org/obo/IDO_0000586 denotes infection
T224 283-288 http://purl.obolibrary.org/obo/NCBITaxon_10239 denotes virus
T225 299-308 http://purl.obolibrary.org/obo/IDO_0000586 denotes infection

LitCovid-sample-PD-FMA

Id Subject Object Predicate Lexical cue fma_id
T400 88-101 Body_part denotes cell membrane http://purl.org/sig/ont/fma/fma63841
T401 88-92 Body_part denotes cell http://purl.org/sig/ont/fma/fma68646

LitCovid-sample-PD-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T334 118-128 Disease denotes SARS-CoV-2 http://purl.obolibrary.org/obo/MONDO_0100096
T335 118-122 Disease denotes SARS http://purl.obolibrary.org/obo/MONDO_0005091
T336 129-138 Disease denotes infection http://purl.obolibrary.org/obo/MONDO_0005550
T337 299-308 Disease denotes infection http://purl.obolibrary.org/obo/MONDO_0005550

LitCovid-sample-PD-HP

Id Subject Object Predicate Lexical cue hp_id
T59 407-427 Phenotype denotes respiratory distress http://purl.obolibrary.org/obo/HP_0002098

LitCovid-PD-HP

Id Subject Object Predicate Lexical cue hp_id
T62 407-427 Phenotype denotes respiratory distress http://purl.obolibrary.org/obo/HP_0002098

LitCovid-PubTator

Id Subject Object Predicate Lexical cue tao:has_database_id
1611 72-76 Gene denotes ACE2 Gene:59272
1612 243-247 Gene denotes ACE2 Gene:59272
1613 364-368 Gene denotes ACE2 Gene:59272
1614 93-101 Gene denotes membrane Gene:43740571
1615 118-138 Disease denotes SARS-CoV-2 infection MESH:C000657245
1616 299-308 Disease denotes infection MESH:D007239
1617 407-427 Disease denotes respiratory distress MESH:D012128

LitCovid-sentences

Id Subject Object Predicate Lexical cue
T356 0-139 Sentence denotes On the whole, there is still uncertainty about the relationship between ACE2 density on cell membrane and the fate of SARS-CoV-2 infection.
T357 140-481 Sentence denotes An interesting speculation which could reconcile the two position comes from the hypothesis that while ACE2 is for sure the entry door for the virus, once the infection has evolved, there is the subsequent downregulation of ACE2, responsible for the precipitating of respiratory distress, as showed also in animal models (Kuba et al., 2005).