PMC:7546716 / 1-432 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T1","span":{"begin":19,"end":30},"obj":"Body_part"},{"id":"T2","span":{"begin":47,"end":51},"obj":"Body_part"},{"id":"T3","span":{"begin":86,"end":94},"obj":"Body_part"},{"id":"T4","span":{"begin":247,"end":255},"obj":"Body_part"},{"id":"T5","span":{"begin":257,"end":268},"obj":"Body_part"},{"id":"T6","span":{"begin":270,"end":272},"obj":"Body_part"}],"attributes":[{"id":"A1","pred":"fma_id","subj":"T1","obj":"http://purl.org/sig/ont/fma/fma86578"},{"id":"A2","pred":"fma_id","subj":"T2","obj":"http://purl.org/sig/ont/fma/fma24994"},{"id":"A3","pred":"fma_id","subj":"T3","obj":"http://purl.org/sig/ont/fma/fma84050"},{"id":"A4","pred":"fma_id","subj":"T4","obj":"http://purl.org/sig/ont/fma/fma84050"},{"id":"A5","pred":"fma_id","subj":"T5","obj":"http://purl.org/sig/ont/fma/fma86578"},{"id":"A6","pred":"fma_id","subj":"T6","obj":"http://purl.org/sig/ont/fma/fma86578"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T1","span":{"begin":47,"end":51},"obj":"Body_part"}],"attributes":[{"id":"A1","pred":"uberon_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/UBERON_0015875"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T1","span":{"begin":55,"end":63},"obj":"Disease"},{"id":"T2","span":{"begin":108,"end":116},"obj":"Disease"},{"id":"T3","span":{"begin":319,"end":327},"obj":"Disease"}],"attributes":[{"id":"A1","pred":"mondo_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"},{"id":"A2","pred":"mondo_id","subj":"T2","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"},{"id":"A3","pred":"mondo_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T1","span":{"begin":207,"end":208},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T2","span":{"begin":281,"end":282},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T1","span":{"begin":270,"end":272},"obj":"Chemical"}],"attributes":[{"id":"A1","pred":"chebi_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/CHEBI_63895"},{"id":"A2","pred":"chebi_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/CHEBI_74072"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-sentences","denotations":[{"id":"T2","span":{"begin":66,"end":74},"obj":"Sentence"},{"id":"T3","span":{"begin":75,"end":189},"obj":"Sentence"},{"id":"T4","span":{"begin":190,"end":409},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}

{"project":"LitCovid-PubTator","denotations":[{"id":"2","span":{"begin":19,"end":33},"obj":"Gene"},{"id":"3","span":{"begin":55,"end":63},"obj":"Disease"},{"id":"23","span":{"begin":257,"end":276},"obj":"Gene"},{"id":"36","span":{"begin":108,"end":116},"obj":"Disease"},{"id":"37","span":{"begin":319,"end":327},"obj":"Disease"}],"attributes":[{"id":"A2","pred":"tao:has_database_id","subj":"2","obj":"Gene:90865"},{"id":"A3","pred":"tao:has_database_id","subj":"3","obj":"MESH:C000657245"},{"id":"A23","pred":"tao:has_database_id","subj":"23","obj":"Gene:90865"},{"id":"A36","pred":"tao:has_database_id","subj":"36","obj":"MESH:C000657245"},{"id":"A37","pred":"tao:has_database_id","subj":"37","obj":"MESH:C000657245"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"mperfect storm: is interleukin-33 the Achilles heel of COVID-19?\n\nAbstract\nThe unique cytokine signature of COVID-19 might provide clues to disease mechanisms and possible future therapies. Here, we propose a pathogenic model in which the alarmin cytokine, interleukin (IL)-33, is a key player in driving all stages of COVID-19 disease (ie, asymptomatic, mild–moderate, severe–critical, and chronic–fibrotic). In susceptible indivi"}