PMC:7534795 / 12774-13237 JSONTXT

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T123","span":{"begin":77,"end":83},"obj":"Disease"},{"id":"T124","span":{"begin":97,"end":105},"obj":"Disease"},{"id":"T125","span":{"begin":241,"end":251},"obj":"Disease"},{"id":"T126","span":{"begin":399,"end":408},"obj":"Disease"}],"attributes":[{"id":"A123","pred":"mondo_id","subj":"T123","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A124","pred":"mondo_id","subj":"T124","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"},{"id":"A125","pred":"mondo_id","subj":"T125","obj":"http://purl.obolibrary.org/obo/MONDO_0000831"},{"id":"A126","pred":"mondo_id","subj":"T126","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T79454","span":{"begin":152,"end":157},"obj":"http://purl.obolibrary.org/obo/NCBITaxon_10239"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T95","span":{"begin":307,"end":310},"obj":"Chemical"}],"attributes":[{"id":"A95","pred":"chebi_id","subj":"T95","obj":"http://purl.obolibrary.org/obo/CHEBI_16750"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T50684","span":{"begin":184,"end":206},"obj":"http://purl.obolibrary.org/obo/GO_0006954"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-sentences","denotations":[{"id":"T80","span":{"begin":122,"end":369},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-PD-HP","denotations":[{"id":"T65","span":{"begin":256,"end":263},"obj":"Phenotype"}],"attributes":[{"id":"A65","pred":"hp_id","subj":"T65","obj":"http://purl.obolibrary.org/obo/HP_0012418"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"2_test","denotations":[{"id":"33031856-32171076-84038834","span":{"begin":363,"end":367},"obj":"32171076"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}

{"project":"LitCovid-PubTator","denotations":[{"id":"521","span":{"begin":410,"end":417},"obj":"Species"},{"id":"524","span":{"begin":66,"end":83},"obj":"Disease"},{"id":"525","span":{"begin":97,"end":105},"obj":"Disease"},{"id":"526","span":{"begin":241,"end":251},"obj":"Disease"},{"id":"527","span":{"begin":256,"end":263},"obj":"Disease"},{"id":"528","span":{"begin":399,"end":408},"obj":"Disease"}],"attributes":[{"id":"A521","pred":"tao:has_database_id","subj":"521","obj":"Tax:9606"},{"id":"A524","pred":"tao:has_database_id","subj":"524","obj":"MESH:D009202"},{"id":"A525","pred":"tao:has_database_id","subj":"525","obj":"MESH:C000657245"},{"id":"A526","pred":"tao:has_database_id","subj":"526","obj":"MESH:D013927"},{"id":"A527","pred":"tao:has_database_id","subj":"527","obj":"MESH:D000860"},{"id":"A528","pred":"tao:has_database_id","subj":"528","obj":"MESH:D007239"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"ID-19 and consequently the exact pathophysiological mechanisms of myocardial injury secondary to COVID-19 remain elusive. However, direct damage by the virus, exaggerated uncontrolled inflammatory responses, instability of coronary plaques, thrombosis and hypoxia have been proposed as possible mechanisms (Guo et al., 2020; Zheng et al., 2020; Zhou, Yu, et al., 2020). Importantly, the severity of infection, patient characteristics and host reaction all partici"}