PMC:7445716 / 2332-2737 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T7","span":{"begin":312,"end":318},"obj":"Body_part"}],"attributes":[{"id":"A7","pred":"fma_id","subj":"T7","obj":"http://purl.org/sig/ont/fma/fma9721"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T3","span":{"begin":312,"end":318},"obj":"Body_part"}],"attributes":[{"id":"A3","pred":"uberon_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/UBERON_0000043"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T33","span":{"begin":2,"end":25},"obj":"Disease"},{"id":"T34","span":{"begin":27,"end":30},"obj":"Disease"},{"id":"T35","span":{"begin":40,"end":43},"obj":"Disease"},{"id":"T36","span":{"begin":175,"end":197},"obj":"Disease"},{"id":"T37","span":{"begin":199,"end":246},"obj":"Disease"},{"id":"T38","span":{"begin":218,"end":246},"obj":"Disease"},{"id":"T39","span":{"begin":232,"end":246},"obj":"Disease"},{"id":"T40","span":{"begin":247,"end":251},"obj":"Disease"},{"id":"T41","span":{"begin":364,"end":367},"obj":"Disease"}],"attributes":[{"id":"A33","pred":"mondo_id","subj":"T33","obj":"http://purl.obolibrary.org/obo/MONDO_0016218"},{"id":"A34","pred":"mondo_id","subj":"T34","obj":"http://purl.obolibrary.org/obo/MONDO_0016218"},{"id":"A35","pred":"mondo_id","subj":"T35","obj":"http://purl.obolibrary.org/obo/MONDO_0016218"},{"id":"A36","pred":"mondo_id","subj":"T36","obj":"http://purl.obolibrary.org/obo/MONDO_0006915"},{"id":"A37","pred":"mondo_id","subj":"T37","obj":"http://purl.obolibrary.org/obo/MONDO_0016218"},{"id":"A38","pred":"mondo_id","subj":"T38","obj":"http://purl.obolibrary.org/obo/MONDO_0003334"},{"id":"A39","pred":"mondo_id","subj":"T39","obj":"http://purl.obolibrary.org/obo/MONDO_0001824"},{"id":"A40","pred":"mondo_id","subj":"T40","obj":"http://purl.obolibrary.org/obo/MONDO_0020347"},{"id":"A41","pred":"mondo_id","subj":"T41","obj":"http://purl.obolibrary.org/obo/MONDO_0016218"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T17","span":{"begin":33,"end":37},"obj":"http://purl.obolibrary.org/obo/CLO_0001417"},{"id":"T18","span":{"begin":312,"end":318},"obj":"http://www.ebi.ac.uk/cellline#tendon"},{"id":"T19","span":{"begin":312,"end":318},"obj":"http://www.ebi.ac.uk/efo/EFO_0000952"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PubTator","denotations":[{"id":"81","span":{"begin":86,"end":103},"obj":"Disease"},{"id":"82","span":{"begin":161,"end":197},"obj":"Disease"},{"id":"83","span":{"begin":218,"end":246},"obj":"Disease"},{"id":"84","span":{"begin":289,"end":297},"obj":"Disease"},{"id":"85","span":{"begin":299,"end":327},"obj":"Disease"},{"id":"86","span":{"begin":333,"end":349},"obj":"Disease"}],"attributes":[{"id":"A81","pred":"tao:has_database_id","subj":"81","obj":"MESH:D010243"},{"id":"A82","pred":"tao:has_database_id","subj":"82","obj":"MESH:D020277"},{"id":"A83","pred":"tao:has_database_id","subj":"83","obj":"MESH:D003711"},{"id":"A84","pred":"tao:has_database_id","subj":"84","obj":"MESH:D018908"},{"id":"A85","pred":"tao:has_database_id","subj":"85","obj":"MESH:D012021"},{"id":"A86","pred":"tao:has_database_id","subj":"86","obj":"MESH:D012678"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-sentences","denotations":[{"id":"T20","span":{"begin":40,"end":109},"obj":"Sentence"},{"id":"T21","span":{"begin":110,"end":350},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PD-HP","denotations":[{"id":"T3","span":{"begin":94,"end":103},"obj":"Phenotype"},{"id":"T4","span":{"begin":161,"end":174},"obj":"Phenotype"},{"id":"T5","span":{"begin":218,"end":231},"obj":"Phenotype"},{"id":"T6","span":{"begin":232,"end":246},"obj":"Phenotype"},{"id":"T7","span":{"begin":299,"end":327},"obj":"Phenotype"}],"attributes":[{"id":"A3","pred":"hp_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/HP_0003470"},{"id":"A4","pred":"hp_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A5","pred":"hp_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A6","pred":"hp_id","subj":"T6","obj":"http://purl.obolibrary.org/obo/HP_0001271"},{"id":"A7","pred":"hp_id","subj":"T7","obj":"http://purl.obolibrary.org/obo/HP_0001284"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

{"project":"LitCovid-PMC-OGER-BB","denotations":[{"id":"T27","span":{"begin":10,"end":11},"obj":"NCBITaxon:10376"},{"id":"T28","span":{"begin":133,"end":139},"obj":"UBERON:0002405"},{"id":"T29","span":{"begin":312,"end":318},"obj":"UBERON:0000043"},{"id":"T30","span":{"begin":319,"end":327},"obj":"GO:0060004"},{"id":"T71449","span":{"begin":10,"end":11},"obj":"NCBITaxon:10376"},{"id":"T6430","span":{"begin":133,"end":139},"obj":"UBERON:0002405"},{"id":"T42852","span":{"begin":312,"end":318},"obj":"UBERON:0000043"},{"id":"T93969","span":{"begin":319,"end":327},"obj":"GO:0060004"}],"text":"f Guillain–Barré Syndrome (GBS) [5–56].\nGBS represents the most common cause of acute flaccid paralysis [57]. The classic form is an immune-mediated acute-onset demyelinating polyradiculoneuropathy (acute inflammatory demyelinating polyneuropathy—AIDP) typically presenting with ascending weakness, loss of deep tendon reflexes, and sensory deficits. Diagnosis of GBS relies on the results of clinical, el"}

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