PMC:7417788 / 55852-56133 JSONTXT

{"project":"LitCovid-PMC-OGER-BB","denotations":[{"id":"T1234","span":{"begin":34,"end":39},"obj":"PR:000016867"},{"id":"T1235","span":{"begin":80,"end":87},"obj":"CHEBI:59132;CHEBI:59132;GO:0019882"},{"id":"T1236","span":{"begin":87,"end":100},"obj":"GO:0019882"},{"id":"T1237","span":{"begin":126,"end":132},"obj":"PR:000010094"},{"id":"T1238","span":{"begin":137,"end":140},"obj":"PR:000003858"},{"id":"T1239","span":{"begin":247,"end":260},"obj":"GO:0065007"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T997","span":{"begin":113,"end":116},"obj":"http://purl.obolibrary.org/obo/CL_0000451"},{"id":"T998","span":{"begin":230,"end":234},"obj":"http://purl.obolibrary.org/obo/CLO_0001185"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T27","span":{"begin":80,"end":87},"obj":"Chemical"}],"attributes":[{"id":"A25457","pred":"chebi_id","subj":"T27","obj":"http://purl.obolibrary.org/obo/CHEBI_59132"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T271","span":{"begin":80,"end":100},"obj":"http://purl.obolibrary.org/obo/GO_0019882"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"LitCovid-sentences","denotations":[{"id":"T288","span":{"begin":0,"end":281},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"2_test","denotations":[{"id":"32780218-29706550-132718722","span":{"begin":24,"end":28},"obj":"29706550"},{"id":"32780218-29706550-132718723","span":{"begin":53,"end":57},"obj":"29706550"},{"id":"32780218-29891535-132718724","span":{"begin":213,"end":217},"obj":"29891535"},{"id":"32780218-29706550-132718725","span":{"begin":230,"end":234},"obj":"29706550"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}

{"project":"LitCovid-PubTator","denotations":[{"id":"1250","span":{"begin":0,"end":7},"obj":"Gene"},{"id":"1251","span":{"begin":34,"end":39},"obj":"Gene"},{"id":"1252","span":{"begin":126,"end":132},"obj":"Gene"},{"id":"1253","span":{"begin":137,"end":140},"obj":"Gene"}],"attributes":[{"id":"A1250","pred":"tao:has_database_id","subj":"1250","obj":"Gene:160364"},{"id":"A1251","pred":"tao:has_database_id","subj":"1251","obj":"Gene:10628"},{"id":"A1252","pred":"tao:has_database_id","subj":"1252","obj":"Gene:378938"},{"id":"A1253","pred":"tao:has_database_id","subj":"1253","obj":"Gene:196"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"CLEC12A (Hutten et al., 2016) and TXNIP (Son et al., 2008) are critical for the antigen-presentation function of DCs, whereas MALAT1 and AHR are critical for tolerogenic DC differentiation (Takenaka and Quintana, 2017; Wu et al., 2018), and their dysregulation hampers DC function."}