PMC:7358770 / 16898-17328
Annnotations
LitCovid-PD-FMA-UBERON
{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T174","span":{"begin":56,"end":60},"obj":"Body_part"},{"id":"T175","span":{"begin":160,"end":166},"obj":"Body_part"},{"id":"T176","span":{"begin":302,"end":306},"obj":"Body_part"},{"id":"T177","span":{"begin":343,"end":351},"obj":"Body_part"}],"attributes":[{"id":"A174","pred":"fma_id","subj":"T174","obj":"http://purl.org/sig/ont/fma/fma7195"},{"id":"A175","pred":"fma_id","subj":"T175","obj":"http://purl.org/sig/ont/fma/fma9637"},{"id":"A176","pred":"fma_id","subj":"T176","obj":"http://purl.org/sig/ont/fma/fma7195"},{"id":"A177","pred":"fma_id","subj":"T177","obj":"http://purl.org/sig/ont/fma/fma82768"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PD-UBERON
{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T7","span":{"begin":56,"end":60},"obj":"Body_part"},{"id":"T8","span":{"begin":160,"end":166},"obj":"Body_part"},{"id":"T9","span":{"begin":302,"end":306},"obj":"Body_part"}],"attributes":[{"id":"A7","pred":"uberon_id","subj":"T7","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"A8","pred":"uberon_id","subj":"T8","obj":"http://purl.obolibrary.org/obo/UBERON_0000479"},{"id":"A9","pred":"uberon_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PubTator
{"project":"LitCovid-PubTator","denotations":[{"id":"564","span":{"begin":48,"end":52},"obj":"Gene"},{"id":"565","span":{"begin":178,"end":182},"obj":"Gene"},{"id":"566","span":{"begin":212,"end":216},"obj":"Gene"},{"id":"567","span":{"begin":56,"end":69},"obj":"Disease"},{"id":"568","span":{"begin":80,"end":86},"obj":"Disease"},{"id":"569","span":{"begin":416,"end":424},"obj":"Disease"}],"attributes":[{"id":"A564","pred":"tao:has_database_id","subj":"564","obj":"Gene:1956"},{"id":"A565","pred":"tao:has_database_id","subj":"565","obj":"Gene:1956"},{"id":"A566","pred":"tao:has_database_id","subj":"566","obj":"Gene:1956"},{"id":"A567","pred":"tao:has_database_id","subj":"567","obj":"MESH:D005355"},{"id":"A568","pred":"tao:has_database_id","subj":"568","obj":"MESH:D014947"},{"id":"A569","pred":"tao:has_database_id","subj":"569","obj":"MESH:D005355"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PD-MONDO
{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T66","span":{"begin":80,"end":86},"obj":"Disease"},{"id":"T67","span":{"begin":117,"end":122},"obj":"Disease"},{"id":"T68","span":{"begin":257,"end":262},"obj":"Disease"},{"id":"T69","span":{"begin":398,"end":403},"obj":"Disease"}],"attributes":[{"id":"A66","pred":"mondo_id","subj":"T66","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A67","pred":"mondo_id","subj":"T67","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A68","pred":"mondo_id","subj":"T68","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A69","pred":"mondo_id","subj":"T69","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PD-CLO
{"project":"LitCovid-PD-CLO","denotations":[{"id":"T157","span":{"begin":56,"end":60},"obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"T158","span":{"begin":56,"end":60},"obj":"http://www.ebi.ac.uk/efo/EFO_0000934"},{"id":"T159","span":{"begin":90,"end":91},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T160","span":{"begin":197,"end":207},"obj":"http://purl.obolibrary.org/obo/CLO_0001658"},{"id":"T161","span":{"begin":291,"end":292},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T162","span":{"begin":302,"end":306},"obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"T163","span":{"begin":302,"end":306},"obj":"http://www.ebi.ac.uk/efo/EFO_0000934"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PD-CHEBI
{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T286","span":{"begin":183,"end":190},"obj":"Chemical"},{"id":"T287","span":{"begin":327,"end":337},"obj":"Chemical"},{"id":"T288","span":{"begin":343,"end":351},"obj":"Chemical"}],"attributes":[{"id":"A286","pred":"chebi_id","subj":"T286","obj":"http://purl.obolibrary.org/obo/CHEBI_52214"},{"id":"A287","pred":"chebi_id","subj":"T287","obj":"http://purl.obolibrary.org/obo/CHEBI_35222"},{"id":"A288","pred":"chebi_id","subj":"T288","obj":"http://purl.obolibrary.org/obo/CHEBI_18186"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PD-GO-BP
{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T22","span":{"begin":48,"end":52},"obj":"http://purl.obolibrary.org/obo/GO_0005006"},{"id":"T23","span":{"begin":117,"end":130},"obj":"http://purl.obolibrary.org/obo/GO_0042060"},{"id":"T24","span":{"begin":178,"end":182},"obj":"http://purl.obolibrary.org/obo/GO_0005006"},{"id":"T25","span":{"begin":212,"end":216},"obj":"http://purl.obolibrary.org/obo/GO_0005006"},{"id":"T26","span":{"begin":257,"end":270},"obj":"http://purl.obolibrary.org/obo/GO_0042060"},{"id":"T27","span":{"begin":398,"end":411},"obj":"http://purl.obolibrary.org/obo/GO_0042060"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
2_test
{"project":"2_test","denotations":[{"id":"32679328-28390872-48281287","span":{"begin":426,"end":428},"obj":"28390872"},{"id":"T95227","span":{"begin":426,"end":428},"obj":"28390872"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-sentences
{"project":"LitCovid-sentences","denotations":[{"id":"T104","span":{"begin":0,"end":70},"obj":"Sentence"},{"id":"T105","span":{"begin":71,"end":225},"obj":"Sentence"},{"id":"T106","span":{"begin":226,"end":309},"obj":"Sentence"},{"id":"T107","span":{"begin":310,"end":430},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
LitCovid-PMC-OGER-BB
{"project":"LitCovid-PMC-OGER-BB","denotations":[{"id":"T460","span":{"begin":56,"end":60},"obj":"UBERON:0002048"},{"id":"T461","span":{"begin":117,"end":130},"obj":"GO:0042060"},{"id":"T462","span":{"begin":160,"end":166},"obj":"UBERON:0000479"},{"id":"T463","span":{"begin":257,"end":270},"obj":"GO:0042060"},{"id":"T464","span":{"begin":302,"end":306},"obj":"UBERON:0002048"},{"id":"T465","span":{"begin":327,"end":337},"obj":"CHEBI:35222;CHEBI:35222"},{"id":"T466","span":{"begin":398,"end":411},"obj":"GO:0042060"},{"id":"T56183","span":{"begin":89,"end":94},"obj":"NCBITaxon:10239"},{"id":"T66872","span":{"begin":95,"end":105},"obj":"UBERON:0001004"},{"id":"T14904","span":{"begin":195,"end":210},"obj":"CHEBI:18154;CHEBI:18154"},{"id":"T54314","span":{"begin":214,"end":225},"obj":"NCBITaxon:11118"},{"id":"T98755","span":{"begin":231,"end":236},"obj":"NCBITaxon:8782"},{"id":"T82764","span":{"begin":237,"end":248},"obj":"NCBITaxon:11118"},{"id":"T30287","span":{"begin":338,"end":343},"obj":"NCBITaxon:8782"},{"id":"T88059","span":{"begin":364,"end":367},"obj":"PR:000003015"},{"id":"T70997","span":{"begin":397,"end":408},"obj":"GO:0006260"},{"id":"T63381","span":{"begin":412,"end":415},"obj":"NCBITaxon:10376"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}
MyTest
{"project":"MyTest","denotations":[{"id":"564","span":{"begin":48,"end":52},"obj":"Gene"},{"id":"565","span":{"begin":178,"end":182},"obj":"Gene"},{"id":"566","span":{"begin":212,"end":216},"obj":"Gene"},{"id":"567","span":{"begin":56,"end":69},"obj":"Disease"},{"id":"568","span":{"begin":80,"end":86},"obj":"Disease"},{"id":"569","span":{"begin":416,"end":424},"obj":"Disease"}],"attributes":[{"id":"A564","pred":"tao:has_database_id","subj":"564","obj":"Gene:1956"},{"id":"A565","pred":"tao:has_database_id","subj":"565","obj":"Gene:1956"},{"id":"A566","pred":"tao:has_database_id","subj":"566","obj":"Gene:1956"},{"id":"A567","pred":"tao:has_database_id","subj":"567","obj":"MESH:D005355"},{"id":"A568","pred":"tao:has_database_id","subj":"568","obj":"MESH:D014947"},{"id":"A569","pred":"tao:has_database_id","subj":"569","obj":"MESH:D005355"}],"namespaces":[{"prefix":"_base","uri":"https://www.uniprot.org/uniprot/testbase"},{"prefix":"UniProtKB","uri":"https://www.uniprot.org/uniprot/"},{"prefix":"uniprot","uri":"https://www.uniprot.org/uniprotkb/"}],"text":"Fig. 5 The illustration about potential role of EGFR in lung fibrosis. Physical injury or a pathogen ① initiates the wound healing response by damaging healthy tissue, releasing EGFR ligands ② and activating the EGFR pathway. This results in an exaggerated wound healing response leading to a fibrotic lung ③. The early use of inhibitors like tyrosine kinase ④ could prevent the normal progress of wound healing and fibrosis [72]."}