PMC:7346000 / 424-666 JSONTXT

{"project":"LitCovid-PubTator","denotations":[{"id":"40","span":{"begin":40,"end":48},"obj":"Disease"},{"id":"41","span":{"begin":65,"end":117},"obj":"Disease"},{"id":"42","span":{"begin":159,"end":182},"obj":"Disease"}],"attributes":[{"id":"A40","pred":"tao:has_database_id","subj":"40","obj":"MESH:C000657245"},{"id":"A41","pred":"tao:has_database_id","subj":"41","obj":"MESH:D008171"},{"id":"A42","pred":"tao:has_database_id","subj":"42","obj":"MESH:D055732"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"ciated molecular patterns during severe COVID-19 results in both pulmonary epithelial damage and inflammatory disease, which are predisposing risk factors for pulmonary aspergillosis. Moreover, collateral effects of host recognition pathways "}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T13","span":{"begin":40,"end":48},"obj":"Disease"},{"id":"T14","span":{"begin":97,"end":117},"obj":"Disease"},{"id":"T15","span":{"begin":169,"end":182},"obj":"Disease"}],"attributes":[{"id":"A13","pred":"mondo_id","subj":"T13","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"},{"id":"A14","pred":"mondo_id","subj":"T14","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"},{"id":"A15","pred":"mondo_id","subj":"T15","obj":"http://purl.obolibrary.org/obo/MONDO_0005657"}],"text":"ciated molecular patterns during severe COVID-19 results in both pulmonary epithelial damage and inflammatory disease, which are predisposing risk factors for pulmonary aspergillosis. Moreover, collateral effects of host recognition pathways "}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T3","span":{"begin":75,"end":85},"obj":"http://purl.obolibrary.org/obo/CL_0000066"}],"text":"ciated molecular patterns during severe COVID-19 results in both pulmonary epithelial damage and inflammatory disease, which are predisposing risk factors for pulmonary aspergillosis. Moreover, collateral effects of host recognition pathways "}