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PMC:7346000 / 0-853 JSONTXT

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LitCovid-PubTator

Id Subject Object Predicate Lexical cue tao:has_database_id
2 0-8 Disease denotes COVID-19 MESH:C000657245
3 20-43 Disease denotes Pulmonary Aspergillosis MESH:D055732
25 256-264 Species denotes patients Tax:9606
28 102-111 Species denotes influenza Tax:11309
33 113-135 Disease denotes coronavirus disease-19 MESH:C000657245
34 137-145 Disease denotes COVID-19 MESH:C000657245
35 160-195 Disease denotes acute respiratory distress syndrome MESH:D012128
36 197-201 Disease denotes ARDS MESH:D012128
37 278-301 Disease denotes pulmonary aspergillosis MESH:D055732
38 320-328 Disease denotes COVID-19 MESH:C000657245
39 340-363 Disease denotes pulmonary aspergillosis MESH:D055732
40 464-472 Disease denotes COVID-19 MESH:C000657245
41 489-541 Disease denotes pulmonary epithelial damage and inflammatory disease MESH:D008171
42 583-606 Disease denotes pulmonary aspergillosis MESH:D055732

LitCovid-PD-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T1 0-8 Disease denotes COVID-19 http://purl.obolibrary.org/obo/MONDO_0100096
T2 30-43 Disease denotes Aspergillosis http://purl.obolibrary.org/obo/MONDO_0005657
T3 45-49 Disease denotes CAPA http://purl.obolibrary.org/obo/MONDO_0007163
T4 102-111 Disease denotes influenza http://purl.obolibrary.org/obo/MONDO_0005812
T5 137-145 Disease denotes COVID-19 http://purl.obolibrary.org/obo/MONDO_0100096
T6 160-195 Disease denotes acute respiratory distress syndrome http://purl.obolibrary.org/obo/MONDO_0006502
T7 166-195 Disease denotes respiratory distress syndrome http://purl.obolibrary.org/obo/MONDO_0009971
T8 197-201 Disease denotes ARDS http://purl.obolibrary.org/obo/MONDO_0006502
T9 288-301 Disease denotes aspergillosis http://purl.obolibrary.org/obo/MONDO_0005657
T10 320-328 Disease denotes COVID-19 http://purl.obolibrary.org/obo/MONDO_0100096
T11 350-363 Disease denotes aspergillosis http://purl.obolibrary.org/obo/MONDO_0005657
T12 365-369 Disease denotes CAPA http://purl.obolibrary.org/obo/MONDO_0007163
T13 464-472 Disease denotes COVID-19 http://purl.obolibrary.org/obo/MONDO_0100096
T14 521-541 Disease denotes inflammatory disease http://purl.obolibrary.org/obo/MONDO_0021166
T15 593-606 Disease denotes aspergillosis http://purl.obolibrary.org/obo/MONDO_0005657
T16 841-845 Disease denotes CAPA http://purl.obolibrary.org/obo/MONDO_0007163

LitCovid-PD-CLO

Id Subject Object Predicate Lexical cue
T1 203-206 http://purl.obolibrary.org/obo/CLO_0051582 denotes has
T2 308-310 http://purl.obolibrary.org/obo/CLO_0001000 denotes 35
T3 499-509 http://purl.obolibrary.org/obo/CL_0000066 denotes epithelial
T4 683-693 http://purl.obolibrary.org/obo/CLO_0001658 denotes activation
T5 750-751 http://purl.obolibrary.org/obo/CLO_0001020 denotes a

LitCovid-PD-CHEBI

Id Subject Object Predicate Lexical cue chebi_id
T1 697-706 Chemical denotes antiviral http://purl.obolibrary.org/obo/CHEBI_22587

LitCovid-PD-GO-BP

Id Subject Object Predicate Lexical cue
T1 814-826 http://purl.obolibrary.org/obo/GO_0009405 denotes pathogenesis

LitCovid-PD-HP

Id Subject Object Predicate Lexical cue hp_id
T1 166-186 Phenotype denotes respiratory distress http://purl.obolibrary.org/obo/HP_0002098

LitCovid-sentences

Id Subject Object Predicate Lexical cue
T1 0-79 Sentence denotes COVID-19 Associated Pulmonary Aspergillosis (CAPA)—From Immunology to Treatment
T2 81-89 Sentence denotes Abstract
T3 90-397 Sentence denotes Like severe influenza, coronavirus disease-19 (COVID-19) resulting in acute respiratory distress syndrome (ARDS) has emerged as an important disease that predisposes patients to secondary pulmonary aspergillosis, with 35 cases of COVID-19 associated pulmonary aspergillosis (CAPA) published until June 2020.
T4 398-607 Sentence denotes The release of danger-associated molecular patterns during severe COVID-19 results in both pulmonary epithelial damage and inflammatory disease, which are predisposing risk factors for pulmonary aspergillosis.
T5 608-827 Sentence denotes Moreover, collateral effects of host recognition pathways required for the activation of antiviral immunity may, paradoxically, contribute to a highly permissive inflammatory environment that favors fungal pathogenesis.