PMC:7321036 / 34059-34499 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T268","span":{"begin":7,"end":12},"obj":"Body_part"},{"id":"T269","span":{"begin":196,"end":201},"obj":"Body_part"},{"id":"T270","span":{"begin":226,"end":231},"obj":"Body_part"},{"id":"T271","span":{"begin":365,"end":383},"obj":"Body_part"}],"attributes":[{"id":"A268","pred":"fma_id","subj":"T268","obj":"http://purl.org/sig/ont/fma/fma67843"},{"id":"A269","pred":"fma_id","subj":"T269","obj":"http://purl.org/sig/ont/fma/fma68646"},{"id":"A270","pred":"fma_id","subj":"T270","obj":"http://purl.org/sig/ont/fma/fma68646"},{"id":"A271","pred":"fma_id","subj":"T271","obj":"http://purl.org/sig/ont/fma/fma84684"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T153","span":{"begin":51,"end":59},"obj":"Disease"},{"id":"T154","span":{"begin":62,"end":71},"obj":"Disease"},{"id":"T155","span":{"begin":412,"end":428},"obj":"Disease"}],"attributes":[{"id":"A153","pred":"mondo_id","subj":"T153","obj":"http://purl.obolibrary.org/obo/MONDO_0005091"},{"id":"A154","pred":"mondo_id","subj":"T154","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A155","pred":"mondo_id","subj":"T155","obj":"http://purl.obolibrary.org/obo/MONDO_0005108"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T296","span":{"begin":47,"end":48},"obj":"http://purl.obolibrary.org/obo/CLO_0001021"},{"id":"T297","span":{"begin":80,"end":81},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T298","span":{"begin":196,"end":201},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T299","span":{"begin":226,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T300","span":{"begin":387,"end":388},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T222","span":{"begin":37,"end":39},"obj":"Chemical"},{"id":"T223","span":{"begin":44,"end":46},"obj":"Chemical"}],"attributes":[{"id":"A222","pred":"chebi_id","subj":"T222","obj":"http://purl.obolibrary.org/obo/CHEBI_27560"},{"id":"A223","pred":"chebi_id","subj":"T223","obj":"http://purl.obolibrary.org/obo/CHEBI_29388"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T223","span":{"begin":412,"end":428},"obj":"http://purl.obolibrary.org/obo/GO_0016032"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-PubTator","denotations":[{"id":"625","span":{"begin":51,"end":71},"obj":"Disease"},{"id":"626","span":{"begin":412,"end":428},"obj":"Disease"}],"attributes":[{"id":"A625","pred":"tao:has_database_id","subj":"625","obj":"MESH:C000657245"},{"id":"A626","pred":"tao:has_database_id","subj":"626","obj":"MESH:D001102"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}

{"project":"LitCovid-sentences","denotations":[{"id":"T224","span":{"begin":51,"end":214},"obj":"Sentence"},{"id":"T225","span":{"begin":215,"end":342},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"ips of actin-rich filopodia (Figures 5B and S3 B). SARS-CoV-2 infection induced a dramatic increase in filopodial protrusions, which were significantly longer and more branched than in uninfected cells (Figure 5D). Uninfected cells also exhibited filopodial protrusions, but their frequency and shape were dramatically different (Figure S3A). Reorganization of the actin cytoskeleton is a common feature of many viral infections and is asso"}