PMC:7309518 / 2078-2851 JSONTXT

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{"target":"https://pubannotation.org/docs/sourcedb/PMC/sourceid/7309518","sourcedb":"PMC","sourceid":"7309518","source_url":"https://www.ncbi.nlm.nih.gov/pmc/7309518","text":"dressed; the role of complement in innate immunity to viral responses and anti-complement therapeutics (i.e. eculizumab) were reviewed.\n\nConclusions\nEmerging data indicate that COVID-19 can trigger not only GBS but other autoimmune neurological diseases necessitating vigilance for early diagnosis and therapy initiation. Although COVID-19 infection, like most other viruses, can potentially worsen patients with pre-existing autoimmunity, there is no evidence that patients with autoimmune neurological diseases stable on common immunotherapies are facing increased risks of infection.\n\nGuillain-Barré syndromes (GBSs) comprise a spectrum of polyneuropathies characterized by acute (within 1–4 weeks) ascending motor weakness, mild or moderate sensory abnormalities, 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