PMC:7200337 / 11461-11692 JSONTXT

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T77","span":{"begin":208,"end":216},"obj":"Disease"}],"attributes":[{"id":"A77","pred":"mondo_id","subj":"T77","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"}],"text":"Taken together, the multiplicity of strategies developed by pathogenic CoVs to escape immune sensing, particularly the IFN-I pathway, suggests a critical role played by the dysregulation of IFN-I response in COVID-19 pathogenicity."}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T46300","span":{"begin":143,"end":144},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"Taken together, the multiplicity of strategies developed by pathogenic CoVs to escape immune sensing, particularly the IFN-I pathway, suggests a critical role played by the dysregulation of IFN-I response in COVID-19 pathogenicity."}

{"project":"LitCovid-PubTator","denotations":[{"id":"425","span":{"begin":71,"end":75},"obj":"Species"},{"id":"431","span":{"begin":208,"end":216},"obj":"Disease"}],"attributes":[{"id":"A425","pred":"tao:has_database_id","subj":"425","obj":"Tax:11118"},{"id":"A431","pred":"tao:has_database_id","subj":"431","obj":"MESH:C000657245"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"Taken together, the multiplicity of strategies developed by pathogenic CoVs to escape immune sensing, particularly the IFN-I pathway, suggests a critical role played by the dysregulation of IFN-I response in COVID-19 pathogenicity."}

{"project":"LitCovid-sentences","denotations":[{"id":"T68","span":{"begin":0,"end":231},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Taken together, the multiplicity of strategies developed by pathogenic CoVs to escape immune sensing, particularly the IFN-I pathway, suggests a critical role played by the dysregulation of IFN-I response in COVID-19 pathogenicity."}