PMC:6796545 / 27820-28811 JSONTXT

{"project":"2_test","denotations":[{"id":"31649699-11427680-41039460","span":{"begin":281,"end":285},"obj":"11427680"},{"id":"31649699-22577183-41039461","span":{"begin":298,"end":302},"obj":"22577183"},{"id":"31649699-25066672-41039462","span":{"begin":324,"end":328},"obj":"25066672"},{"id":"31649699-29659946-41039463","span":{"begin":342,"end":346},"obj":"29659946"},{"id":"31649699-30154805-41039464","span":{"begin":967,"end":971},"obj":"30154805"},{"id":"31649699-29659946-41039465","span":{"begin":985,"end":989},"obj":"29659946"}],"text":"Ethylene is also a key regulator of the expression of sex-controlling genes. Treatment with exogenous ethylene at an appropriate concentration increased the transcription of CsACS1, CsACS2, CsACS11, CmACS11, and CmACS7 and downregulated that of CsWIP1 and CmWIP1 (Yamasaki et al., 2001; Li et al., 2012; Switzenberg et al., 2014; Tao et al., 2018). Endogenous ethylene produced by a first expressed sex-specific gene might also act with other sex-controlling genes, which is used to explain the interacting phenomenon among them (see below). A hypothesis was proposed that ethylene mediated the interaction among different sex-controlling genes, including (to date, at least) CsACS2, CsACS11, CmACS11, CmACS7, CsWIP1, and CmWIP1. Cloning of ethylene signaling factors, CsERF110/CmERF110 and CsERF31, which directly combine with the promoters of CsACS11/CmACS11 and CsACS2 to activate their expression, respectively, supplied evidence for this hypothesis (Pan et al., 2018; Tao et al., 2018)."}

{"project":"CucurLitBase-testset_Gene","denotations":[{"id":"T27","span":{"begin":174,"end":180},"obj":"CsACS1"},{"id":"T28","span":{"begin":182,"end":188},"obj":"CsACS2"},{"id":"T29","span":{"begin":676,"end":682},"obj":"CsACS2"},{"id":"T30","span":{"begin":791,"end":798},"obj":"CsERF31"},{"id":"T31","span":{"begin":865,"end":871},"obj":"CsACS2"}],"text":"Ethylene is also a key regulator of the expression of sex-controlling genes. Treatment with exogenous ethylene at an appropriate concentration increased the transcription of CsACS1, CsACS2, CsACS11, CmACS11, and CmACS7 and downregulated that of CsWIP1 and CmWIP1 (Yamasaki et al., 2001; Li et al., 2012; Switzenberg et al., 2014; Tao et al., 2018). Endogenous ethylene produced by a first expressed sex-specific gene might also act with other sex-controlling genes, which is used to explain the interacting phenomenon among them (see below). A hypothesis was proposed that ethylene mediated the interaction among different sex-controlling genes, including (to date, at least) CsACS2, CsACS11, CmACS11, CmACS7, CsWIP1, and CmWIP1. Cloning of ethylene signaling factors, CsERF110/CmERF110 and CsERF31, which directly combine with the promoters of CsACS11/CmACS11 and CsACS2 to activate their expression, respectively, supplied evidence for this hypothesis (Pan et al., 2018; Tao et al., 2018)."}

{"project":"CucurLitBase-testset_BFT","denotations":[{"id":"T71","span":{"begin":174,"end":180},"obj":"CsACS1"},{"id":"T72","span":{"begin":182,"end":188},"obj":"CsACS2"},{"id":"T73","span":{"begin":676,"end":682},"obj":"CsACS2"},{"id":"T74","span":{"begin":791,"end":798},"obj":"CsERF31"},{"id":"T75","span":{"begin":865,"end":871},"obj":"CsACS2"}],"text":"Ethylene is also a key regulator of the expression of sex-controlling genes. Treatment with exogenous ethylene at an appropriate concentration increased the transcription of CsACS1, CsACS2, CsACS11, CmACS11, and CmACS7 and downregulated that of CsWIP1 and CmWIP1 (Yamasaki et al., 2001; Li et al., 2012; Switzenberg et al., 2014; Tao et al., 2018). Endogenous ethylene produced by a first expressed sex-specific gene might also act with other sex-controlling genes, which is used to explain the interacting phenomenon among them (see below). A hypothesis was proposed that ethylene mediated the interaction among different sex-controlling genes, including (to date, at least) CsACS2, CsACS11, CmACS11, CmACS7, CsWIP1, and CmWIP1. Cloning of ethylene signaling factors, CsERF110/CmERF110 and CsERF31, which directly combine with the promoters of CsACS11/CmACS11 and CsACS2 to activate their expression, respectively, supplied evidence for this hypothesis (Pan et al., 2018; Tao et al., 2018)."}