PMC:3320587 / 36-721
Annnotations
pmc-enju-pas
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HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway\nNFAT5 Modulation of MTb-Induced HIV-1 Replication\n\nAbstract\nTuberculosis (TB) disease in HIV co-infected patients contributes to increased mortality by activating innate and adaptive immune signaling cascades that stimulate HIV-1 replication, leading to an increase in viral load. Here, we demonstrate that silencing of the expression of the transcription factor nuclear factor of activated T cells 5 (NFAT5) by RNA interference (RNAi) inhibits Mycobacterium tuberculosis (MTb)-stimulated HIV-1 replication in co-infected macrophages. We show that NFAT5 gene and protein expression are s"}
bionlp-st-ge-2016-test-proteins
{"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T85","span":{"begin":58,"end":63},"obj":"Protein"},{"id":"T86","span":{"begin":461,"end":498},"obj":"Protein"},{"id":"T87","span":{"begin":500,"end":505},"obj":"Protein"},{"id":"T88","span":{"begin":646,"end":651},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"osis-Dependent HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway\nNFAT5 Modulation of MTb-Induced HIV-1 Replication\n\nAbstract\nTuberculosis (TB) disease in HIV co-infected patients contributes to increased mortality by activating innate and adaptive immune signaling cascades that stimulate HIV-1 replication, leading to an increase in viral load. Here, we demonstrate that silencing of the expression of the transcription factor nuclear factor of activated T cells 5 (NFAT5) by RNA interference (RNAi) inhibits Mycobacterium tuberculosis (MTb)-stimulated HIV-1 replication in co-infected macrophages. We show that NFAT5 gene and protein expression are s"}
bionlp-st-ge-2016-uniprot
{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T412","span":{"begin":500,"end":505},"obj":"http://www.uniprot.org/uniprot/O94916"},{"id":"T413","span":{"begin":646,"end":651},"obj":"http://www.uniprot.org/uniprot/O94916"},{"id":"T411","span":{"begin":58,"end":63},"obj":"http://www.uniprot.org/uniprot/O94916"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"osis-Dependent HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway\nNFAT5 Modulation of MTb-Induced HIV-1 Replication\n\nAbstract\nTuberculosis (TB) disease in HIV co-infected patients contributes to increased mortality by activating innate and adaptive immune signaling cascades that stimulate HIV-1 replication, leading to an increase in viral load. Here, we demonstrate that silencing of the expression of the transcription factor nuclear factor of activated T cells 5 (NFAT5) by RNA interference (RNAi) inhibits Mycobacterium tuberculosis (MTb)-stimulated HIV-1 replication in co-infected macrophages. We show that NFAT5 gene and protein expression are s"}
GO-BP
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GO-CC
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sentences
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ICD10
{"project":"ICD10","denotations":[{"id":"T116","span":{"begin":557,"end":569},"obj":"http://purl.bioontology.org/ontology/ICD10/A15-A19.9"}],"text":"osis-Dependent HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway\nNFAT5 Modulation of MTb-Induced HIV-1 Replication\n\nAbstract\nTuberculosis (TB) disease in HIV co-infected patients contributes to increased mortality by activating innate and adaptive immune signaling cascades that stimulate HIV-1 replication, leading to an increase in viral load. Here, we demonstrate that silencing of the expression of the transcription factor nuclear factor of activated T cells 5 (NFAT5) by RNA interference (RNAi) inhibits Mycobacterium tuberculosis (MTb)-stimulated HIV-1 replication in co-infected macrophages. We show that NFAT5 gene and protein expression are s"}
simple1
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BioNLP16_DUT
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BioNLP16_Messiy
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DLUT931
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bionlp-st-ge-2016-test-ihmc
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bionlp-st-ge-2016-spacy-parsed
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bionlp-st-ge-2016-test-tees
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testone
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test3
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