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{"project":"GO-CC","denotations":[{"id":"T5683","span":{"begin":404,"end":411},"obj":"http://purl.obolibrary.org/obo/GO_0005634"},{"id":"T5682","span":{"begin":38,"end":47},"obj":"http://purl.obolibrary.org/obo/GO_0005737"}],"text":"NF-κB proteins are sequestered in the cytoplasm as latent complexes by inhibitory proteins referred to as inhibitor of NF-κB (IκB) proteins, which prevent NF-κB nuclear translocation and DNA binding [19]. Whereas the majority of IκBs (IκBα, IκBβ, IκBε, p105 (also known as NF-κB1), p100 (also known as NF-κB2)) serve as inhibitors of NF-κB, IκBξ and Bcl-3 instead potentiate NF-κB transactivation in the nucleus. p100 and p105 are precursors of the p52 and p50 NF-κB subunits, respectively. There are two unique NF-κB signalling pathways, termed canonical (or classical) and noncanonical (or alternative) NF-κB pathways."}

{"project":"sentences","denotations":[{"id":"T4239","span":{"begin":491,"end":620},"obj":"Sentence"},{"id":"T4238","span":{"begin":205,"end":490},"obj":"Sentence"},{"id":"T4237","span":{"begin":0,"end":204},"obj":"Sentence"},{"id":"T39","span":{"begin":0,"end":204},"obj":"Sentence"},{"id":"T40","span":{"begin":205,"end":490},"obj":"Sentence"},{"id":"T41","span":{"begin":491,"end":620},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"NF-κB proteins are sequestered in the cytoplasm as latent complexes by inhibitory proteins referred to as inhibitor of NF-κB (IκB) proteins, which prevent NF-κB nuclear translocation and DNA binding [19]. Whereas the majority of IκBs (IκBα, IκBβ, IκBε, p105 (also known as NF-κB1), p100 (also known as NF-κB2)) serve as inhibitors of NF-κB, IκBξ and Bcl-3 instead potentiate NF-κB transactivation in the nucleus. p100 and p105 are precursors of the p52 and p50 NF-κB subunits, respectively. There are two unique NF-κB signalling pathways, termed canonical (or classical) and noncanonical (or alternative) NF-κB pathways."}

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{"project":"BioNLP16_DUT","denotations":[{"id":"T7831","span":{"begin":457,"end":460},"obj":"Protein"},{"id":"T7830","span":{"begin":449,"end":452},"obj":"Protein"},{"id":"T7829","span":{"begin":422,"end":426},"obj":"Protein"},{"id":"T7828","span":{"begin":413,"end":417},"obj":"Protein"},{"id":"T7827","span":{"begin":350,"end":355},"obj":"Protein"},{"id":"T7826","span":{"begin":341,"end":345},"obj":"Protein"},{"id":"T7825","span":{"begin":302,"end":308},"obj":"Protein"},{"id":"T7824","span":{"begin":282,"end":286},"obj":"Protein"},{"id":"T7823","span":{"begin":273,"end":279},"obj":"Protein"},{"id":"T7822","span":{"begin":253,"end":257},"obj":"Protein"},{"id":"T7821","span":{"begin":247,"end":251},"obj":"Protein"},{"id":"T7820","span":{"begin":241,"end":245},"obj":"Protein"},{"id":"T7819","span":{"begin":235,"end":239},"obj":"Protein"}],"text":"NF-κB proteins are sequestered in the cytoplasm as latent complexes by inhibitory proteins referred to as inhibitor of NF-κB (IκB) proteins, which prevent NF-κB nuclear translocation and DNA binding [19]. Whereas the majority of IκBs (IκBα, IκBβ, IκBε, p105 (also known as NF-κB1), p100 (also known as NF-κB2)) serve as inhibitors of NF-κB, IκBξ and Bcl-3 instead potentiate NF-κB transactivation in the nucleus. p100 and p105 are precursors of the p52 and p50 NF-κB subunits, respectively. There are two unique NF-κB signalling pathways, termed canonical (or classical) and noncanonical (or alternative) NF-κB pathways."}

{"project":"BioNLP16_Messiy","denotations":[{"id":"T6954","span":{"begin":457,"end":460},"obj":"Protein"},{"id":"T6953","span":{"begin":449,"end":452},"obj":"Protein"},{"id":"T6952","span":{"begin":422,"end":426},"obj":"Protein"},{"id":"T6951","span":{"begin":413,"end":417},"obj":"Protein"},{"id":"T6950","span":{"begin":350,"end":355},"obj":"Protein"},{"id":"T6949","span":{"begin":341,"end":345},"obj":"Protein"},{"id":"T6948","span":{"begin":302,"end":308},"obj":"Protein"},{"id":"T6947","span":{"begin":282,"end":286},"obj":"Protein"},{"id":"T6946","span":{"begin":273,"end":279},"obj":"Protein"},{"id":"T6945","span":{"begin":253,"end":257},"obj":"Protein"},{"id":"T6944","span":{"begin":247,"end":251},"obj":"Protein"},{"id":"T6943","span":{"begin":241,"end":245},"obj":"Protein"},{"id":"T6942","span":{"begin":235,"end":239},"obj":"Protein"}],"text":"NF-κB proteins are sequestered in the cytoplasm as latent complexes by inhibitory proteins referred to as inhibitor of NF-κB (IκB) proteins, which prevent NF-κB nuclear translocation and DNA binding [19]. Whereas the majority of IκBs (IκBα, IκBβ, IκBε, p105 (also known as NF-κB1), p100 (also known as NF-κB2)) serve as inhibitors of NF-κB, IκBξ and Bcl-3 instead potentiate NF-κB transactivation in the nucleus. p100 and p105 are precursors of the p52 and p50 NF-κB subunits, respectively. There are two unique NF-κB signalling pathways, termed canonical (or classical) and noncanonical (or alternative) NF-κB pathways."}

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