PMC:3218875 / 1840-2096 JSONTXT

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    testone

    {"project":"testone","denotations":[{"id":"T271","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T270","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T269","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T268","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T267","span":{"begin":44,"end":47},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    bionlp-st-ge-2016-spacy-parsed

    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of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    DLUT931

    {"project":"DLUT931","denotations":[{"id":"T1394","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T1393","span":{"begin":44,"end":47},"obj":"Protein"},{"id":"T1385","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T1384","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T1383","span":{"begin":55,"end":59},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    bionlp-st-ge-2016-uniprot

    {"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T1276","span":{"begin":55,"end":59},"obj":"http://www.uniprot.org/uniprot/P05231"},{"id":"T1275","span":{"begin":195,"end":198},"obj":"http://www.uniprot.org/uniprot/P01375"},{"id":"T1274","span":{"begin":44,"end":47},"obj":"http://www.uniprot.org/uniprot/P01375"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    bionlp-st-ge-2016-test-proteins

    {"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T367","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T366","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T365","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T364","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T363","span":{"begin":44,"end":47},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    pmc-enju-pas

    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of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    GO-BP

    {"project":"GO-BP","denotations":[{"id":"T794","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0051001"},{"id":"T793","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0050999"},{"id":"T792","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0051000"},{"id":"T791","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0004517"},{"id":"T790","span":{"begin":67,"end":76},"obj":"http://purl.obolibrary.org/obo/GO_0016701"},{"id":"T782","span":{"begin":235,"end":254},"obj":"http://purl.obolibrary.org/obo/GO_0051092"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    GO-MF

    {"project":"GO-MF","denotations":[{"id":"T809","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0050998"},{"id":"T808","span":{"begin":102,"end":123},"obj":"http://purl.obolibrary.org/obo/GO_0004517"},{"id":"T807","span":{"begin":67,"end":76},"obj":"http://purl.obolibrary.org/obo/GO_0016701"},{"id":"T806","span":{"begin":55,"end":59},"obj":"http://purl.obolibrary.org/obo/GO_0005138"},{"id":"T805","span":{"begin":203,"end":207},"obj":"http://purl.obolibrary.org/obo/GO_0005149"},{"id":"T804","span":{"begin":49,"end":53},"obj":"http://purl.obolibrary.org/obo/GO_0005149"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    simple1

    {"project":"simple1","denotations":[{"id":"T825","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T824","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T823","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T822","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T821","span":{"begin":44,"end":47},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    BioNLP16_DUT

    {"project":"BioNLP16_DUT","denotations":[{"id":"T1518","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T1517","span":{"begin":44,"end":47},"obj":"Protein"},{"id":"T1509","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T1508","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T1507","span":{"begin":55,"end":59},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    BioNLP16_Messiy

    {"project":"BioNLP16_Messiy","denotations":[{"id":"T1291","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T1290","span":{"begin":44,"end":47},"obj":"Protein"},{"id":"T1282","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T1281","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T1280","span":{"begin":55,"end":59},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    bionlp-st-ge-2016-test-ihmc

    {"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T1576","span":{"begin":92,"end":124},"obj":"Protein"},{"id":"T1571","span":{"begin":28,"end":43},"obj":"Protein"},{"id":"T1569","span":{"begin":49,"end":53},"obj":"Protein"},{"id":"T1567","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T1564","span":{"begin":223,"end":254},"obj":"Entity"},{"id":"T1561","span":{"begin":175,"end":180},"obj":"Protein"},{"id":"T1558","span":{"begin":203,"end":207},"obj":"Protein"},{"id":"T1553","span":{"begin":44,"end":54},"obj":"Protein"},{"id":"T1548","span":{"begin":25,"end":135},"obj":"Protein"},{"id":"T1542","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T1530","span":{"begin":249,"end":254},"obj":"Protein"},{"id":"T1529","span":{"begin":55,"end":79},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    bionlp-st-ge-2016-test-tees

    {"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T1313","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T1312","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T1311","span":{"begin":49,"end":53},"obj":"Protein"},{"id":"T1310","span":{"begin":44,"end":47},"obj":"Protein"},{"id":"T1322","span":{"begin":92,"end":101},"obj":"Positive_regulation"},{"id":"T1315","span":{"begin":136,"end":154},"obj":"Protein"},{"id":"T1314","span":{"begin":102,"end":123},"obj":"Protein"},{"id":"T1363","span":{"begin":235,"end":245},"obj":"Positive_regulation"},{"id":"T1362","span":{"begin":235,"end":245},"obj":"Positive_regulation"},{"id":"T1361","span":{"begin":249,"end":254},"obj":"Protein"},{"id":"T1360","span":{"begin":203,"end":207},"obj":"Protein"},{"id":"T1359","span":{"begin":195,"end":198},"obj":"Protein"}],"relations":[{"id":"R1065","pred":"themeOf","subj":"T1314","obj":"T1322"},{"id":"R1092","pred":"themeOf","subj":"T1361","obj":"T1362"},{"id":"R1093","pred":"themeOf","subj":"T1361","obj":"T1363"},{"id":"R1094","pred":"Cause","subj":"T1362","obj":"T1359"},{"id":"R1095","pred":"Cause","subj":"T1363","obj":"T1360"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}

    test3

    {"project":"test3","denotations":[{"id":"T314","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T313","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T312","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T311","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T310","span":{"begin":44,"end":47},"obj":"Protein"},{"id":"T293","span":{"begin":195,"end":198},"obj":"Protein"},{"id":"T292","span":{"begin":92,"end":123},"obj":"Protein"},{"id":"T291","span":{"begin":61,"end":78},"obj":"Protein"},{"id":"T290","span":{"begin":55,"end":59},"obj":"Protein"},{"id":"T289","span":{"begin":44,"end":47},"obj":"Protein"}],"text":"ession of a large number of genes - such as TNF, IL-1, IL-6, cyclo-oxygenase-2, chemokines, inducible nitric oxide synthase, and matrix metalloproteinases - that are involved in RA. In addition, TNF and IL-1 are themselves very potent activators of NF-κB ("}