PMC:2948162 / 1655-2313 JSONTXT

{"project":"0_colil","denotations":[{"id":"20577881-912839-54375","span":{"begin":190,"end":191},"obj":"912839"},{"id":"20577881-4543365-54376","span":{"begin":652,"end":653},"obj":"4543365"},{"id":"20577881-4140198-54377","span":{"begin":655,"end":656},"obj":"4140198"}],"text":"Coronary occlusion that persists for more than 40 min leads to irreversible myocardial damage that starts in the endocardium and progresses towards the epicardium as a wavefront phenomenon [1]. Although timely reperfusion salvages myocardium and reduces mortality, successful restoration of epicardial artery patency after prolonged occlusion does not always lead to adequate reperfusion at the microvascular level. Reperfused acute myocardial infarction (AMI) is characterised by myocyte swelling due to osmotic overload, intracellular calcium overload, myocyte hypercontraction, microvascular obstruction (MVO) and intramyocardial haemorrhage (IMH) [2, 3]."}

{"project":"2_test","denotations":[{"id":"20577881-912839-29362307","span":{"begin":190,"end":191},"obj":"912839"},{"id":"20577881-4543365-29362308","span":{"begin":652,"end":653},"obj":"4543365"},{"id":"20577881-4140198-29362309","span":{"begin":655,"end":656},"obj":"4140198"}],"text":"Coronary occlusion that persists for more than 40 min leads to irreversible myocardial damage that starts in the endocardium and progresses towards the epicardium as a wavefront phenomenon [1]. Although timely reperfusion salvages myocardium and reduces mortality, successful restoration of epicardial artery patency after prolonged occlusion does not always lead to adequate reperfusion at the microvascular level. Reperfused acute myocardial infarction (AMI) is characterised by myocyte swelling due to osmotic overload, intracellular calcium overload, myocyte hypercontraction, microvascular obstruction (MVO) and intramyocardial haemorrhage (IMH) [2, 3]."}

{"project":"TEST0","denotations":[{"id":"20577881-190-195-54375","span":{"begin":190,"end":191},"obj":"[\"912839\"]"},{"id":"20577881-236-241-54376","span":{"begin":652,"end":653},"obj":"[\"4543365\"]"},{"id":"20577881-228-233-54377","span":{"begin":655,"end":656},"obj":"[\"4140198\"]"}],"text":"Coronary occlusion that persists for more than 40 min leads to irreversible myocardial damage that starts in the endocardium and progresses towards the epicardium as a wavefront phenomenon [1]. Although timely reperfusion salvages myocardium and reduces mortality, successful restoration of epicardial artery patency after prolonged occlusion does not always lead to adequate reperfusion at the microvascular level. Reperfused acute myocardial infarction (AMI) is characterised by myocyte swelling due to osmotic overload, intracellular calcium overload, myocyte hypercontraction, microvascular obstruction (MVO) and intramyocardial haemorrhage (IMH) [2, 3]."}

{"project":"MyTest","denotations":[{"id":"20577881-912839-29362307","span":{"begin":190,"end":191},"obj":"912839"},{"id":"20577881-4543365-29362308","span":{"begin":652,"end":653},"obj":"4543365"},{"id":"20577881-4140198-29362309","span":{"begin":655,"end":656},"obj":"4140198"}],"namespaces":[{"prefix":"_base","uri":"https://www.uniprot.org/uniprot/testbase"},{"prefix":"UniProtKB","uri":"https://www.uniprot.org/uniprot/"},{"prefix":"uniprot","uri":"https://www.uniprot.org/uniprotkb/"}],"text":"Coronary occlusion that persists for more than 40 min leads to irreversible myocardial damage that starts in the endocardium and progresses towards the epicardium as a wavefront phenomenon [1]. Although timely reperfusion salvages myocardium and reduces mortality, successful restoration of epicardial artery patency after prolonged occlusion does not always lead to adequate reperfusion at the microvascular level. Reperfused acute myocardial infarction (AMI) is characterised by myocyte swelling due to osmotic overload, intracellular calcium overload, myocyte hypercontraction, microvascular obstruction (MVO) and intramyocardial haemorrhage (IMH) [2, 3]."}