PMC:2427286 / 17647-19174 JSONTXT

{"target":"https://pubannotation.org/docs/sourcedb/PMC/sourceid/2427286","sourcedb":"PMC","sourceid":"2427286","source_url":"https://www.ncbi.nlm.nih.gov/pmc/2427286","text":"These observations, together with the historical information, led us to formulate three specific hypotheses: that many J∗(xJ2) chromosomes were introduced into Lebanese Muslims by the Muslim expansion from the Arabian Peninsula; that some I and R1b chromosomes were introduced into Lebanese Christians by immigrating European Christians, perhaps during the time of the Crusades; and that additional R1b chromosomes were introduced into Lebanese Muslims during the Ottoman expansion. We do not, of course, imply that these migrations carried only these haplogroups; obviously, they would have involved populations containing multiple haplogroups. The signal of migration, however, should be most readily detected in the highly differentiated haplogroups. J∗(xJ2) was found to be much more frequent in Lebanese Muslims than in Lebanese non-Muslims (25% vs. 15%, p \u003c 0.0001). The combined I + R1b frequency was higher in Lebanese Christians than in Lebanese non-Christians (16% vs. 10%, p = 0.01), as were both of the individual haplogroups (I: 5.8% vs. 4.0%, p = 0.21; R1b 10% vs. 6.3%, p = 0.03), although the difference for haplogroup I alone did not reach statistical significance. The R1b frequency was, however, significantly lower in Lebanese Muslims than in Lebanese non-Muslims (4.7% vs. 11%, p = 0.0005). The hypotheses of male-mediated gene flow accompanying the earlier Muslim and Crusader migrations are therefore supported, but our data provide no evidence for a differential genetic impact of the Ottoman expansion.","tracks":[]}