PMC:2195754 / 12617-13525 JSONTXT

{"target":"http://pubannotation.org/docs/sourcedb/PMC/sourceid/2195754","sourcedb":"PMC","sourceid":"2195754","source_url":"https://www.ncbi.nlm.nih.gov/pmc/2195754","text":"Analysis of Non-HLs.\nThe six IκBα exons were amplified as above from DNA of peripheral blood lymphocytes (\u003e80% tumor cells) of chronic lymphocytic leukemia patients (CLL1–6, 9, 10, 13–15 16) and from the Burkitt's lymphoma cell lines BL31, BL36, BL41, BL60, BL74 (obtained from G. Lenoir, International Agency for Research on Cancer [IARC], Lyon, France), Namalwa (from M. Pawlita, Deutsches Krebsforschungszentrum [DKFZ], Heidelberg, Germany), mutu (from M. Falk, Institut für Klinische Molekularbiologie und Tumorgenetik, München, Germany), BJAB (from E. Kieff, Brigham and Women's Hospital, Boston, MA), and Ramos (from M. Neuberger, Medical Research Council, Cambridge, UK). To confirm a germline polymorphism in BL60 (351G→C on one allele, leading to a Gly→Arg exchange at position 74 in the IκBα protein), exon 1 was amplified from IARC277, a lymphoblastoid cell line established from the same patient.","divisions":[{"label":"Title","span":{"begin":0,"end":20}}],"tracks":[]}