PMC:1472690 / 295-741 JSONTXT

{"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T302","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T303","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T304","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T305","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T306","span":{"begin":420,"end":426},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T433","span":{"begin":304,"end":311},"obj":"http://www.uniprot.org/uniprot/P06886"},{"id":"T434","span":{"begin":420,"end":426},"obj":"http://www.uniprot.org/uniprot/P06886"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"UBERON-AE","denotations":[{"id":"T187","span":{"begin":151,"end":156},"obj":"http://purl.obolibrary.org/obo/UBERON_0000178"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"GO-BP","denotations":[{"id":"T207","span":{"begin":111,"end":119},"obj":"http://purl.obolibrary.org/obo/GO_0007349"},{"id":"T208","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0002449"},{"id":"T209","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0002456"},{"id":"T210","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0019724"},{"id":"T211","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0002228"},{"id":"T212","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0002443"},{"id":"T213","span":{"begin":157,"end":178},"obj":"http://purl.obolibrary.org/obo/GO_0001909"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"GO-MF","denotations":[{"id":"T348","span":{"begin":191,"end":205},"obj":"http://purl.obolibrary.org/obo/GO_0001872"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"GO-CC","denotations":[{"id":"T356","span":{"begin":164,"end":169},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"sentences","denotations":[{"id":"T192","span":{"begin":317,"end":324},"obj":"Sentence"},{"id":"T6","span":{"begin":317,"end":324},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"ICD10","denotations":[{"id":"T333","span":{"begin":277,"end":297},"obj":"http://purl.bioontology.org/ontology/ICD10/A48.3"},{"id":"T334","span":{"begin":283,"end":288},"obj":"http://purl.bioontology.org/ontology/ICD10/R57.9"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"simple1","denotations":[{"id":"T395","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T396","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T397","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T398","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T399","span":{"begin":420,"end":426},"obj":"Protein"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"BioNLP16_DUT","denotations":[{"id":"T861","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T827","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T828","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T832","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T833","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T834","span":{"begin":420,"end":426},"obj":"Protein"}],"relations":[{"id":"R341","pred":"themeOf","subj":"T832","obj":"T861"},{"id":"R342","pred":"themeOf","subj":"T833","obj":"T861"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"BioNLP16_Messiy","denotations":[{"id":"T488","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T453","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T454","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T458","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T459","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T460","span":{"begin":420,"end":426},"obj":"Protein"}],"relations":[{"id":"R250","pred":"themeOf","subj":"T458","obj":"T488"},{"id":"R251","pred":"themeOf","subj":"T459","obj":"T488"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"DLUT931","denotations":[{"id":"T606","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T573","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T574","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T578","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T579","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T580","span":{"begin":420,"end":426},"obj":"Protein"}],"relations":[{"id":"R301","pred":"themeOf","subj":"T579","obj":"T606"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T761","span":{"begin":212,"end":218},"obj":"Entity"},{"id":"T765","span":{"begin":397,"end":427},"obj":"Protein"},{"id":"T767","span":{"begin":420,"end":426},"obj":"Protein"},{"id":"T774","span":{"begin":277,"end":314},"obj":"Protein"},{"id":"T777","span":{"begin":157,"end":205},"obj":"Entity"},{"id":"T778","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T790","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T792","span":{"begin":246,"end":273},"obj":"Entity"},{"id":"T800","span":{"begin":413,"end":416},"obj":"Entity"},{"id":"T804","span":{"begin":269,"end":272},"obj":"Entity"},{"id":"T740","span":{"begin":212,"end":314},"obj":"Entity"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T514","span":{"begin":350,"end":371},"obj":"Protein"},{"id":"T521","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T522","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T523","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T515","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T516","span":{"begin":397,"end":401},"obj":"Protein"}],"relations":[{"id":"R263","pred":"themeOf","subj":"T514","obj":"T521"},{"id":"R264","pred":"themeOf","subj":"T515","obj":"T522"},{"id":"R265","pred":"themeOf","subj":"T516","obj":"T523"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"testone","denotations":[{"id":"T38","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T39","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T40","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T41","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T42","span":{"begin":420,"end":426},"obj":"Protein"}],"relations":[{"id":"R1","pred":"equivalentTo","subj":"T39","obj":"T38"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}

{"project":"test3","denotations":[{"id":"T100","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T101","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T102","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T103","span":{"begin":420,"end":426},"obj":"Protein"},{"id":"T99","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T135","span":{"begin":277,"end":305},"obj":"Protein"},{"id":"T136","span":{"begin":307,"end":313},"obj":"Protein"},{"id":"T137","span":{"begin":336,"end":346},"obj":"Positive_regulation"},{"id":"T138","span":{"begin":373,"end":392},"obj":"Protein"},{"id":"T139","span":{"begin":397,"end":401},"obj":"Protein"},{"id":"T140","span":{"begin":420,"end":426},"obj":"Protein"}],"relations":[{"id":"R41","pred":"equivalentTo","subj":"T136","obj":"T135"},{"id":"R42","pred":"themeOf","subj":"T138","obj":"T137"},{"id":"R43","pred":"themeOf","subj":"T139","obj":"T137"},{"id":"R44","pred":"themeOf","subj":"T140","obj":"T137"}],"text":"ng a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1).\n\nResults\nDespite an activation of nuclear factor (NF)κB, NFinterleukin(IL)-6 and NFAT similar to LPS or TSST-1, we observed no sig"}