PMC:1382200 / 798-1038 JSONTXT

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{"project":"craft-ca-core-dev","denotations":[{"id":"T554","span":{"begin":32,"end":37},"obj":"GO:0016265"},{"id":"T555","span":{"begin":41,"end":46},"obj":"PR:000005022"},{"id":"T556","span":{"begin":50,"end":54},"obj":"NCBITaxon:10088"},{"id":"T557","span":{"begin":167,"end":172},"obj":"NCBITaxon:10088"},{"id":"T558","span":{"begin":173,"end":178},"obj":"PR:000005015"},{"id":"T559","span":{"begin":179,"end":183},"obj":"SO:0000704"},{"id":"T560","span":{"begin":187,"end":196},"obj":"UBERON:0000922"},{"id":"T561","span":{"begin":187,"end":208},"obj":"CL:0002322"}],"text":"f m-calpain was responsible for death of Capn4-/- mice.\n\nResults\nTo distinguish between the alternatives described above, we deleted an essential coding region in the mouse Capn2 gene in embryonic stems cells and transmitted this mutant all"}

{"project":"craft-ca-core-ex-dev","denotations":[{"id":"T623","span":{"begin":2,"end":11},"obj":"GO_PR_EXT:calpain_2_complex"},{"id":"T624","span":{"begin":32,"end":37},"obj":"GO:0016265"},{"id":"T625","span":{"begin":41,"end":46},"obj":"PR_EXT:000005022"},{"id":"T626","span":{"begin":46,"end":47},"obj":"SO_EXT:sequence_nullness_or_absence"},{"id":"T627","span":{"begin":48,"end":49},"obj":"SO_EXT:sequence_nullness_or_absence"},{"id":"T628","span":{"begin":50,"end":54},"obj":"NCBITaxon:10088"},{"id":"T629","span":{"begin":125,"end":132},"obj":"SO_EXT:sequence_deletion_process"},{"id":"T630","span":{"begin":146,"end":159},"obj":"SO_EXT:coding_sequence"},{"id":"T631","span":{"begin":167,"end":172},"obj":"NCBITaxon:10088"},{"id":"T632","span":{"begin":173,"end":178},"obj":"PR_EXT:000005015"},{"id":"T633","span":{"begin":179,"end":183},"obj":"SO_EXT:0000704"},{"id":"T634","span":{"begin":187,"end":196},"obj":"UBERON:0000922"},{"id":"T635","span":{"begin":187,"end":208},"obj":"CL:0002322"},{"id":"T636","span":{"begin":203,"end":208},"obj":"CL_GO_EXT:cell"},{"id":"T637","span":{"begin":230,"end":236},"obj":"SO_EXT:sequence_altered_entity_or_alteration_process"}],"text":"f m-calpain was responsible for death of Capn4-/- mice.\n\nResults\nTo distinguish between the alternatives described above, we deleted an essential coding region in the mouse Capn2 gene in embryonic stems cells and transmitted this mutant all"}