PMC:1064895 / 22004-22704 JSONTXT

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RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"bionlp-st-ge-2016-coref","denotations":[{"id":"T19442","span":{"begin":345,"end":357},"obj":"Antecedent"},{"id":"T19441","span":{"begin":336,"end":340},"obj":"Antecedent"},{"id":"T19440","span":{"begin":299,"end":323},"obj":"Anaphor"},{"id":"T19439","span":{"begin":244,"end":249},"obj":"Antecedent"},{"id":"T19438","span":{"begin":235,"end":239},"obj":"Antecedent"},{"id":"T19437","span":{"begin":201,"end":226},"obj":"Anaphor"}],"relations":[{"id":"R10471","pred":"boundBy","subj":"T19437","obj":"T19438"},{"id":"R10472","pred":"boundBy","subj":"T19437","obj":"T19439"},{"id":"R10473","pred":"boundBy","subj":"T19440","obj":"T19441"},{"id":"R10474","pred":"boundBy","subj":"T19440","obj":"T19442"}],"namespaces":[{"prefix":"_base","uri":"https://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T19460","span":{"begin":590,"end":595},"obj":"Protein"},{"id":"T19459","span":{"begin":345,"end":357},"obj":"Protein"},{"id":"T19458","span":{"begin":336,"end":340},"obj":"Protein"},{"id":"T19457","span":{"begin":244,"end":249},"obj":"Protein"},{"id":"T19456","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T19455","span":{"begin":0,"end":5},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T19969","span":{"begin":345,"end":357},"obj":"http://www.uniprot.org/uniprot/P01579"},{"id":"T19968","span":{"begin":336,"end":340},"obj":"http://www.uniprot.org/uniprot/P05112"},{"id":"T19965","span":{"begin":244,"end":249},"obj":"http://www.uniprot.org/uniprot/P01375"},{"id":"T19963","span":{"begin":244,"end":247},"obj":"http://www.uniprot.org/uniprot/P01375"},{"id":"T19932","span":{"begin":590,"end":595},"obj":"http://www.uniprot.org/uniprot/Q16552"},{"id":"T19931","span":{"begin":0,"end":5},"obj":"http://www.uniprot.org/uniprot/Q16552"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"GO-BP","denotations":[{"id":"T18173","span":{"begin":494,"end":506},"obj":"http://purl.obolibrary.org/obo/GO_0006954"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

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was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T19625","span":{"begin":345,"end":357},"obj":"Protein"},{"id":"T19624","span":{"begin":336,"end":340},"obj":"Protein"},{"id":"T19623","span":{"begin":244,"end":249},"obj":"Protein"},{"id":"T19622","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T19621","span":{"begin":0,"end":5},"obj":"Protein"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}

{"project":"testone","denotations":[{"id":"T16903","span":{"begin":590,"end":595},"obj":"Protein"},{"id":"T16902","span":{"begin":345,"end":357},"obj":"Protein"},{"id":"T16901","span":{"begin":336,"end":340},"obj":"Protein"},{"id":"T16900","span":{"begin":244,"end":249},"obj":"Protein"},{"id":"T16899","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T16898","span":{"begin":0,"end":5},"obj":"Protein"}],"text":"IL-17 was first described as a T cell product with proinflammatory properties [5,22]. RA is characterized by hyperplasia of synovial lining cells and an intense infiltration by mononuclear cells [23]. Proinflammatory cytokines such as IL-1 and TNF-α are abundant in rheumatoid synovium, whereas the T cell-derived cytokines, especially IL-4 and interferon-γ, have often proved difficult to detect in RA synovium [24]. Although T cells may have a role in the augmentation of rheumatoid synovial inflammation, the lack of T cell-derived cytokines has limited its importance. In this respect, IL-17 is appealing because it has been described as a T cell-derived cytokine with proinflammatory properties."}