PMC:101377 / 4550-4933 JSONTXT

{"target":"http://pubannotation.org/docs/sourcedb/PMC/sourceid/101377","sourcedb":"PMC","sourceid":"101377","source_url":"https://www.ncbi.nlm.nih.gov/pmc/101377","text":"It is worth noting that also genetic factors unlinked to the beta globin cluster, favour the Hb F synthesis in the post-fetal life [4]. Particularly interesting for its frequency is the polimorphic single-base substitution C→T at Ggamma -158, recognizable for the creation of a XmnI restriction site, that appears to cause an increase of Hb F expecially during erythropoietic stress.","tracks":[]}