CORD-19:a44cd4fe9a7c450cf0e3e0fe955d43f824bb3ebe / 5933-6268 JSONTXT

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T36","span":{"begin":0,"end":335},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"In order to further understand the biological role of CCL2 expression in a CNS inflammatory disease such as TMEV-IDD, we generated a transgenic mouse line where astrocytes constitutively expressed CCL2 at low levels (Huang et al, 2002) under control of the human glial fibrillary acidic protein (hGFAP) promoter (Brenner et al, 1994) ."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T36","span":{"begin":0,"end":335},"obj":"Sentence"}],"text":"In order to further understand the biological role of CCL2 expression in a CNS inflammatory disease such as TMEV-IDD, we generated a transgenic mouse line where astrocytes constitutively expressed CCL2 at low levels (Huang et al, 2002) under control of the human glial fibrillary acidic protein (hGFAP) promoter (Brenner et al, 1994) ."}

{"project":"CORD-19-PD-UBERON","denotations":[{"id":"T31","span":{"begin":75,"end":78},"obj":"Body_part"}],"attributes":[{"id":"A31","pred":"uberon_id","subj":"T31","obj":"http://purl.obolibrary.org/obo/UBERON_0001017"}],"text":"In order to further understand the biological role of CCL2 expression in a CNS inflammatory disease such as TMEV-IDD, we generated a transgenic mouse line where astrocytes constitutively expressed CCL2 at low levels (Huang et al, 2002) under control of the human glial fibrillary acidic protein (hGFAP) promoter (Brenner et al, 1994) ."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T32","span":{"begin":79,"end":99},"obj":"Disease"},{"id":"T33","span":{"begin":113,"end":116},"obj":"Disease"},{"id":"T64900","span":{"begin":79,"end":99},"obj":"Disease"},{"id":"T31","span":{"begin":113,"end":116},"obj":"Disease"}],"attributes":[{"id":"A32","pred":"mondo_id","subj":"T32","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"},{"id":"A33","pred":"mondo_id","subj":"T33","obj":"http://purl.obolibrary.org/obo/MONDO_0011385"},{"id":"A34","pred":"mondo_id","subj":"T33","obj":"http://purl.obolibrary.org/obo/MONDO_0044339"},{"id":"A45243","pred":"mondo_id","subj":"T64900","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"},{"id":"A49625","pred":"mondo_id","subj":"T31","obj":"http://purl.obolibrary.org/obo/MONDO_0011385"},{"id":"A20598","pred":"mondo_id","subj":"T31","obj":"http://purl.obolibrary.org/obo/MONDO_0044339"}],"text":"In order to further understand the biological role of CCL2 expression in a CNS inflammatory disease such as TMEV-IDD, we generated a transgenic mouse line where astrocytes constitutively expressed CCL2 at low levels (Huang et al, 2002) under control of the human glial fibrillary acidic protein (hGFAP) promoter (Brenner et al, 1994) ."}