CORD-19:954e351a7d8b5d4c275b9af0b053ce983d334547 / 67477-67714 JSONTXT

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T98","span":{"begin":0,"end":237},"obj":"Sentence"}],"text":"132, 134 Although neutrophils and mast cells mediate many of the pathophysiologic effects of the complement cascade, the membrane attack complex may have a primary role in altered vascular permeability during ischemia-reperfusion injury."}

{"project":"CORD-19-Sentences","denotations":[{"id":"T7919","span":{"begin":0,"end":237},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"132, 134 Although neutrophils and mast cells mediate many of the pathophysiologic effects of the complement cascade, the membrane attack complex may have a primary role in altered vascular permeability during ischemia-reperfusion injury."}

{"project":"Epistemic_Statements","denotations":[{"id":"T196","span":{"begin":0,"end":237},"obj":"Epistemic_statement"}],"text":"132, 134 Although neutrophils and mast cells mediate many of the pathophysiologic effects of the complement cascade, the membrane attack complex may have a primary role in altered vascular permeability during ischemia-reperfusion injury."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T218","span":{"begin":209,"end":236},"obj":"Disease"},{"id":"T219","span":{"begin":209,"end":217},"obj":"Disease"},{"id":"T220","span":{"begin":230,"end":236},"obj":"Disease"}],"attributes":[{"id":"A218","pred":"mondo_id","subj":"T218","obj":"http://purl.obolibrary.org/obo/MONDO_0005203"},{"id":"A219","pred":"mondo_id","subj":"T219","obj":"http://purl.obolibrary.org/obo/MONDO_0005053"},{"id":"A220","pred":"mondo_id","subj":"T220","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"}],"text":"132, 134 Although neutrophils and mast cells mediate many of the pathophysiologic effects of the complement cascade, the membrane attack complex may have a primary role in altered vascular permeability during ischemia-reperfusion injury."}