CORD-19:932bcdd2f6c6d487386e8bf983e4874387771be1 / 855783-856021 JSONTXT

{"project":"CORD-19-Sentences","denotations":[{"id":"T37569","span":{"begin":0,"end":238},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"In conclusion, AE-mediated down-regulation of IRF-1 and ERK pathways, with subsequent inhibition of iNOS transcription and NO synthesis in glioma cells, might have important implications for potential therapeutic use of this antraquinone."}

{"project":"Epistemic_Statements","denotations":[{"id":"T1712","span":{"begin":0,"end":238},"obj":"Epistemic_statement"}],"text":"In conclusion, AE-mediated down-regulation of IRF-1 and ERK pathways, with subsequent inhibition of iNOS transcription and NO synthesis in glioma cells, might have important implications for potential therapeutic use of this antraquinone."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T359","span":{"begin":0,"end":238},"obj":"Sentence"}],"text":"In conclusion, AE-mediated down-regulation of IRF-1 and ERK pathways, with subsequent inhibition of iNOS transcription and NO synthesis in glioma cells, might have important implications for potential therapeutic use of this antraquinone."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T1767","span":{"begin":139,"end":145},"obj":"Phenotype"}],"attributes":[{"id":"A1767","pred":"hp_id","subj":"T1767","obj":"http://purl.obolibrary.org/obo/HP_0009733"}],"text":"In conclusion, AE-mediated down-regulation of IRF-1 and ERK pathways, with subsequent inhibition of iNOS transcription and NO synthesis in glioma cells, might have important implications for potential therapeutic use of this antraquinone."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T5255","span":{"begin":139,"end":145},"obj":"Disease"},{"id":"T17232","span":{"begin":139,"end":145},"obj":"Disease"}],"attributes":[{"id":"A5255","pred":"mondo_id","subj":"T5255","obj":"http://purl.obolibrary.org/obo/MONDO_0021042"},{"id":"A75490","pred":"mondo_id","subj":"T17232","obj":"http://purl.obolibrary.org/obo/MONDO_0021042"}],"text":"In conclusion, AE-mediated down-regulation of IRF-1 and ERK pathways, with subsequent inhibition of iNOS transcription and NO synthesis in glioma cells, might have important implications for potential therapeutic use of this antraquinone."}