CORD-19:932bcdd2f6c6d487386e8bf983e4874387771be1 / 366662-366874 JSONTXT

{"project":"CORD-19-Sentences","denotations":[{"id":"T90865","span":{"begin":0,"end":212},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}

{"project":"Epistemic_Statements","denotations":[{"id":"T781","span":{"begin":0,"end":212},"obj":"Epistemic_statement"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T257","span":{"begin":0,"end":212},"obj":"Sentence"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T647","span":{"begin":84,"end":87},"obj":"Phenotype"}],"attributes":[{"id":"A647","pred":"hp_id","subj":"T647","obj":"http://purl.obolibrary.org/obo/HP_0002383"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}

{"project":"CORD-19-PD-UBERON","denotations":[{"id":"T1230","span":{"begin":202,"end":205},"obj":"Body_part"}],"attributes":[{"id":"A1230","pred":"uberon_id","subj":"T1230","obj":"http://purl.obolibrary.org/obo/UBERON_0001017"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T2252","span":{"begin":56,"end":64},"obj":"Disease"},{"id":"T2253","span":{"begin":65,"end":82},"obj":"Disease"},{"id":"T2254","span":{"begin":84,"end":87},"obj":"Disease"},{"id":"T2222","span":{"begin":56,"end":64},"obj":"Disease"},{"id":"T17629","span":{"begin":65,"end":82},"obj":"Disease"},{"id":"T2224","span":{"begin":84,"end":87},"obj":"Disease"}],"attributes":[{"id":"A2252","pred":"mondo_id","subj":"T2252","obj":"http://purl.obolibrary.org/obo/MONDO_0004980"},{"id":"A2253","pred":"mondo_id","subj":"T2253","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A2254","pred":"mondo_id","subj":"T2254","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A2255","pred":"mondo_id","subj":"T2254","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A2256","pred":"mondo_id","subj":"T2254","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"},{"id":"A40129","pred":"mondo_id","subj":"T2222","obj":"http://purl.obolibrary.org/obo/MONDO_0004980"},{"id":"A17109","pred":"mondo_id","subj":"T17629","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A72902","pred":"mondo_id","subj":"T2224","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A88517","pred":"mondo_id","subj":"T2224","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A38785","pred":"mondo_id","subj":"T2224","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"}],"text":"Despite many claims for neuroprotection in experimental allergic encephalomyelitis (EAE), the data often indicate that the majority of neuroprotective effects occur secondary to inhibition of leukocyte CNS entry."}