CORD-19:68a7101a90454172c91785d8c352f776a82df5d4 / 635476-635590 JSONTXT

{"project":"CORD-19-Sentences","denotations":[{"id":"T50513","span":{"begin":0,"end":114},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Remarkably, mice lacking the receptors to both IFN-a/b and IFN-g (IFN-a/b/gR −/− ) developed a milder form of EAE."}

{"project":"Epistemic_Statements","denotations":[{"id":"T1380","span":{"begin":0,"end":114},"obj":"Epistemic_statement"}],"text":"Remarkably, mice lacking the receptors to both IFN-a/b and IFN-g (IFN-a/b/gR −/− ) developed a milder form of EAE."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T260","span":{"begin":0,"end":114},"obj":"Sentence"}],"text":"Remarkably, mice lacking the receptors to both IFN-a/b and IFN-g (IFN-a/b/gR −/− ) developed a milder form of EAE."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T1361","span":{"begin":110,"end":113},"obj":"Phenotype"}],"attributes":[{"id":"A1361","pred":"hp_id","subj":"T1361","obj":"http://purl.obolibrary.org/obo/HP_0002383"}],"text":"Remarkably, mice lacking the receptors to both IFN-a/b and IFN-g (IFN-a/b/gR −/− ) developed a milder form of EAE."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T3969","span":{"begin":110,"end":113},"obj":"Disease"},{"id":"T3947","span":{"begin":110,"end":113},"obj":"Disease"}],"attributes":[{"id":"A3969","pred":"mondo_id","subj":"T3969","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"},{"id":"A3970","pred":"mondo_id","subj":"T3969","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A3971","pred":"mondo_id","subj":"T3969","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A42855","pred":"mondo_id","subj":"T3947","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"},{"id":"A49537","pred":"mondo_id","subj":"T3947","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A47579","pred":"mondo_id","subj":"T3947","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"}],"text":"Remarkably, mice lacking the receptors to both IFN-a/b and IFN-g (IFN-a/b/gR −/− ) developed a milder form of EAE."}