CORD-19:68a7101a90454172c91785d8c352f776a82df5d4 / 319611-319841 JSONTXT

{"project":"CORD-19-Sentences","denotations":[{"id":"T31898","span":{"begin":0,"end":230},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}

{"project":"Epistemic_Statements","denotations":[{"id":"T685","span":{"begin":0,"end":230},"obj":"Epistemic_statement"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T139","span":{"begin":0,"end":230},"obj":"Sentence"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T723","span":{"begin":191,"end":194},"obj":"Phenotype"}],"attributes":[{"id":"A723","pred":"hp_id","subj":"T723","obj":"http://purl.obolibrary.org/obo/HP_0002383"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}

{"project":"CORD-19-PD-UBERON","denotations":[{"id":"T1283","span":{"begin":37,"end":40},"obj":"Body_part"},{"id":"T1284","span":{"begin":184,"end":186},"obj":"Body_part"}],"attributes":[{"id":"A1283","pred":"uberon_id","subj":"T1283","obj":"http://purl.obolibrary.org/obo/UBERON_0001017"},{"id":"A1284","pred":"uberon_id","subj":"T1284","obj":"http://purl.obolibrary.org/obo/UBERON_0001877"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T2108","span":{"begin":41,"end":53},"obj":"Disease"},{"id":"T2109","span":{"begin":184,"end":186},"obj":"Disease"},{"id":"T2110","span":{"begin":191,"end":194},"obj":"Disease"},{"id":"T86271","span":{"begin":41,"end":53},"obj":"Disease"},{"id":"T27009","span":{"begin":184,"end":186},"obj":"Disease"},{"id":"T2091","span":{"begin":191,"end":194},"obj":"Disease"}],"attributes":[{"id":"A2108","pred":"mondo_id","subj":"T2108","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"},{"id":"A2109","pred":"mondo_id","subj":"T2109","obj":"http://purl.obolibrary.org/obo/MONDO_0005301"},{"id":"A2110","pred":"mondo_id","subj":"T2110","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"},{"id":"A2111","pred":"mondo_id","subj":"T2110","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A2112","pred":"mondo_id","subj":"T2110","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"},{"id":"A82399","pred":"mondo_id","subj":"T86271","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"},{"id":"A48622","pred":"mondo_id","subj":"T27009","obj":"http://purl.obolibrary.org/obo/MONDO_0005301"},{"id":"A31824","pred":"mondo_id","subj":"T2091","obj":"http://purl.obolibrary.org/obo/MONDO_0005134"},{"id":"A46948","pred":"mondo_id","subj":"T2091","obj":"http://purl.obolibrary.org/obo/MONDO_0019956"},{"id":"A13676","pred":"mondo_id","subj":"T2091","obj":"http://purl.obolibrary.org/obo/MONDO_0005156"}],"text":"To assess the contribution of MIF to CNS inflammation, primary microglial cultures were treated with increasing doses of rMIF, which upregulated inflammatory mediators associated with MS and EAE: IL-1, IL-6, TNF-a, iNOS, and CCL2."}