
CORD-19:4abc50cfc81265083d4cb2aed0a493bb704d0b5b / 10853-11187
Annnotations
CORD-19-PD-MONDO
{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T69","span":{"begin":49,"end":58},"obj":"Disease"},{"id":"T80055","span":{"begin":49,"end":58},"obj":"Disease"}],"attributes":[{"id":"A69","pred":"mondo_id","subj":"T69","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A75900","pred":"mondo_id","subj":"T80055","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"}],"text":"This can be partially explained by the fact that infection with live-attenuated RABV initially stimulates expression of neuronal cytokines, such as CXCL10, which further attract infiltration of inflammatory cells into CNS and boost induction of proinflammatory chemokines/cytokines, such as MIP-1α and interferon-γ (IFN-γ) in the CNS."}
CORD-19-Sentences
{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T57","span":{"begin":0,"end":334},"obj":"Sentence"},{"id":"T90425","span":{"begin":0,"end":334},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"This can be partially explained by the fact that infection with live-attenuated RABV initially stimulates expression of neuronal cytokines, such as CXCL10, which further attract infiltration of inflammatory cells into CNS and boost induction of proinflammatory chemokines/cytokines, such as MIP-1α and interferon-γ (IFN-γ) in the CNS."}
CORD-19-PD-UBERON
{"project":"CORD-19-PD-UBERON","denotations":[{"id":"T16","span":{"begin":218,"end":221},"obj":"Body_part"},{"id":"T17","span":{"begin":330,"end":333},"obj":"Body_part"}],"attributes":[{"id":"A16","pred":"uberon_id","subj":"T16","obj":"http://purl.obolibrary.org/obo/UBERON_0001017"},{"id":"A17","pred":"uberon_id","subj":"T17","obj":"http://purl.obolibrary.org/obo/UBERON_0001017"}],"text":"This can be partially explained by the fact that infection with live-attenuated RABV initially stimulates expression of neuronal cytokines, such as CXCL10, which further attract infiltration of inflammatory cells into CNS and boost induction of proinflammatory chemokines/cytokines, such as MIP-1α and interferon-γ (IFN-γ) in the CNS."}