CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 7676-7965 JSONTXT

Annnotations TAB JSON ListView MergeView

    CORD-19_Custom_license_subset

    {"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T44","span":{"begin":0,"end":289},"obj":"Sentence"}],"text":"Finally, changes in NAA associated with MS may reflect defects or dysregulation of axonal energy-storage mechanisms, as NAA-derived metabolic products such as acetyl coenzyme A may function to meet the high energy demands of axonal electrochemical conduction (Mehta and Namboodiri, 1995) ."}

    CORD-19-Sentences

    {"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T44","span":{"begin":0,"end":289},"obj":"Sentence"},{"id":"TextSentencer_T44","span":{"begin":0,"end":289},"obj":"Sentence"},{"id":"T47562","span":{"begin":0,"end":289},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Finally, changes in NAA associated with MS may reflect defects or dysregulation of axonal energy-storage mechanisms, as NAA-derived metabolic products such as acetyl coenzyme A may function to meet the high energy demands of axonal electrochemical conduction (Mehta and Namboodiri, 1995) ."}

    Epistemic_Statements

    {"project":"Epistemic_Statements","denotations":[{"id":"T23","span":{"begin":0,"end":289},"obj":"Epistemic_statement"}],"text":"Finally, changes in NAA associated with MS may reflect defects or dysregulation of axonal energy-storage mechanisms, as NAA-derived metabolic products such as acetyl coenzyme A may function to meet the high energy demands of axonal electrochemical conduction (Mehta and Namboodiri, 1995) ."}

    CORD-19-PD-MONDO

    {"project":"CORD-19-PD-MONDO","denotations":[{"id":"T25","span":{"begin":40,"end":42},"obj":"Disease"}],"attributes":[{"id":"A25","pred":"mondo_id","subj":"T25","obj":"http://purl.obolibrary.org/obo/MONDO_0005301"}],"text":"Finally, changes in NAA associated with MS may reflect defects or dysregulation of axonal energy-storage mechanisms, as NAA-derived metabolic products such as acetyl coenzyme A may function to meet the high energy demands of axonal electrochemical conduction (Mehta and Namboodiri, 1995) ."}