CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 57089-57415 JSONTXT

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T44","span":{"begin":0,"end":326},"obj":"Sentence"}],"text":"Treatment with β-methylcyclodextrin, a cholesterol-chelating compound that effectively destroys lipid raft structure, prevented rHIgM22-induced survival signaling, suggesting that perhaps redistribution or aggregation of lipid raft domains is necessary for antibody-mediated signal transduction (Howe et al., 2004) (Fig. 20) ."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T286","span":{"begin":0,"end":326},"obj":"Sentence"},{"id":"TextSentencer_T286","span":{"begin":0,"end":326},"obj":"Sentence"},{"id":"T85652","span":{"begin":0,"end":326},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Treatment with β-methylcyclodextrin, a cholesterol-chelating compound that effectively destroys lipid raft structure, prevented rHIgM22-induced survival signaling, suggesting that perhaps redistribution or aggregation of lipid raft domains is necessary for antibody-mediated signal transduction (Howe et al., 2004) (Fig. 20) ."}

{"project":"Epistemic_Statements","denotations":[{"id":"T126","span":{"begin":0,"end":320},"obj":"Epistemic_statement"}],"text":"Treatment with β-methylcyclodextrin, a cholesterol-chelating compound that effectively destroys lipid raft structure, prevented rHIgM22-induced survival signaling, suggesting that perhaps redistribution or aggregation of lipid raft domains is necessary for antibody-mediated signal transduction (Howe et al., 2004) (Fig. 20) ."}