CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 52098-52446 JSONTXT

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CORD-19_Custom_license_subset

Id Subject Object Predicate Lexical cue
T20 0-348 Sentence denotes These data support the conclusion that the delayed response in oligodendrocytes is dependent on Ca 2+ entry through AMPA receptor Ca 2+ channels, whereas the immediate response in astrocytes is dependent on Ca 2+ release from endoplasmic reticulum stores, likely through PLC-mediated generation of IP 3 and gating of IP 3sensitive calcium channels.

CORD-19-Sentences

Id Subject Object Predicate Lexical cue
TextSentencer_T262 0-348 Sentence denotes These data support the conclusion that the delayed response in oligodendrocytes is dependent on Ca 2+ entry through AMPA receptor Ca 2+ channels, whereas the immediate response in astrocytes is dependent on Ca 2+ release from endoplasmic reticulum stores, likely through PLC-mediated generation of IP 3 and gating of IP 3sensitive calcium channels.
TextSentencer_T262 0-348 Sentence denotes These data support the conclusion that the delayed response in oligodendrocytes is dependent on Ca 2+ entry through AMPA receptor Ca 2+ channels, whereas the immediate response in astrocytes is dependent on Ca 2+ release from endoplasmic reticulum stores, likely through PLC-mediated generation of IP 3 and gating of IP 3sensitive calcium channels.
T85614 0-348 Sentence denotes These data support the conclusion that the delayed response in oligodendrocytes is dependent on Ca 2+ entry through AMPA receptor Ca 2+ channels, whereas the immediate response in astrocytes is dependent on Ca 2+ release from endoplasmic reticulum stores, likely through PLC-mediated generation of IP 3 and gating of IP 3sensitive calcium channels.

Epistemic_Statements

Id Subject Object Predicate Lexical cue
T119 0-348 Epistemic_statement denotes These data support the conclusion that the delayed response in oligodendrocytes is dependent on Ca 2+ entry through AMPA receptor Ca 2+ channels, whereas the immediate response in astrocytes is dependent on Ca 2+ release from endoplasmic reticulum stores, likely through PLC-mediated generation of IP 3 and gating of IP 3sensitive calcium channels.