CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 33124-33493 JSONTXT

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T169","span":{"begin":0,"end":369},"obj":"Sentence"}],"text":"In addition, we found that both IFNγ −/− and IFNγ +/+ mice of resistant MHC H-2 b haplotype cleared Theiler's virus infection from spinal motor neurons, but whereas IFNγ +/+ H-2 b mice eventually cleared virus from the spinal cord white matter, IFNγ −/− H-2 b mice developed viral persistence in glial cells of the white matter and consequent spinal cord demyelination."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T169","span":{"begin":0,"end":369},"obj":"Sentence"},{"id":"TextSentencer_T169","span":{"begin":0,"end":369},"obj":"Sentence"},{"id":"T47572","span":{"begin":0,"end":369},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"In addition, we found that both IFNγ −/− and IFNγ +/+ mice of resistant MHC H-2 b haplotype cleared Theiler's virus infection from spinal motor neurons, but whereas IFNγ +/+ H-2 b mice eventually cleared virus from the spinal cord white matter, IFNγ −/− H-2 b mice developed viral persistence in glial cells of the white matter and consequent spinal cord demyelination."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T115","span":{"begin":110,"end":125},"obj":"Disease"},{"id":"T116","span":{"begin":116,"end":125},"obj":"Disease"}],"attributes":[{"id":"A115","pred":"mondo_id","subj":"T115","obj":"http://purl.obolibrary.org/obo/MONDO_0005108"},{"id":"A116","pred":"mondo_id","subj":"T116","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"}],"text":"In addition, we found that both IFNγ −/− and IFNγ +/+ mice of resistant MHC H-2 b haplotype cleared Theiler's virus infection from spinal motor neurons, but whereas IFNγ +/+ H-2 b mice eventually cleared virus from the spinal cord white matter, IFNγ −/− H-2 b mice developed viral persistence in glial cells of the white matter and consequent spinal cord demyelination."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T75","span":{"begin":355,"end":368},"obj":"Phenotype"}],"attributes":[{"id":"A75","pred":"hp_id","subj":"T75","obj":"http://purl.obolibrary.org/obo/HP_0011096"}],"text":"In addition, we found that both IFNγ −/− and IFNγ +/+ mice of resistant MHC H-2 b haplotype cleared Theiler's virus infection from spinal motor neurons, but whereas IFNγ +/+ H-2 b mice eventually cleared virus from the spinal cord white matter, IFNγ −/− H-2 b mice developed viral persistence in glial cells of the white matter and consequent spinal cord demyelination."}

{"project":"CORD-19-PD-UBERON","denotations":[{"id":"T74","span":{"begin":219,"end":230},"obj":"Body_part"},{"id":"T75","span":{"begin":231,"end":243},"obj":"Body_part"},{"id":"T76","span":{"begin":231,"end":236},"obj":"Body_part"},{"id":"T77","span":{"begin":315,"end":327},"obj":"Body_part"},{"id":"T78","span":{"begin":315,"end":320},"obj":"Body_part"},{"id":"T79","span":{"begin":343,"end":354},"obj":"Body_part"}],"attributes":[{"id":"A74","pred":"uberon_id","subj":"T74","obj":"http://purl.obolibrary.org/obo/UBERON_0002240"},{"id":"A75","pred":"uberon_id","subj":"T75","obj":"http://purl.obolibrary.org/obo/UBERON_0002316"},{"id":"A76","pred":"uberon_id","subj":"T76","obj":"http://purl.obolibrary.org/obo/UBERON_0002316"},{"id":"A77","pred":"uberon_id","subj":"T77","obj":"http://purl.obolibrary.org/obo/UBERON_0002316"},{"id":"A78","pred":"uberon_id","subj":"T78","obj":"http://purl.obolibrary.org/obo/UBERON_0002316"},{"id":"A79","pred":"uberon_id","subj":"T79","obj":"http://purl.obolibrary.org/obo/UBERON_0002240"}],"text":"In addition, we found that both IFNγ −/− and IFNγ +/+ mice of resistant MHC H-2 b haplotype cleared Theiler's virus infection from spinal motor neurons, but whereas IFNγ +/+ H-2 b mice eventually cleared virus from the spinal cord white matter, IFNγ −/− H-2 b mice developed viral persistence in glial cells of the white matter and consequent spinal cord demyelination."}