CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 29365-29706 JSONTXT

Annnotations TAB JSON ListView MergeView

CORD-19_Custom_license_subset

Id Subject Object Predicate Lexical cue
T151 0-341 Sentence denotes Although cultured CD4 + and CD8 + T-cells can engage both the perforin and the Fas/FasL pathways, in vivo experiments suggest that MHC class I-dependent killing is dominated by the perforin pathway, whereas MHC class II-dependent elimination is almost entirely mediated by Fas/FasL interactions (Graubert et al., 1997; Schulz et al., 1995) .

CORD-19-Sentences

Id Subject Object Predicate Lexical cue
TextSentencer_T151 0-341 Sentence denotes Although cultured CD4 + and CD8 + T-cells can engage both the perforin and the Fas/FasL pathways, in vivo experiments suggest that MHC class I-dependent killing is dominated by the perforin pathway, whereas MHC class II-dependent elimination is almost entirely mediated by Fas/FasL interactions (Graubert et al., 1997; Schulz et al., 1995) .
TextSentencer_T151 0-341 Sentence denotes Although cultured CD4 + and CD8 + T-cells can engage both the perforin and the Fas/FasL pathways, in vivo experiments suggest that MHC class I-dependent killing is dominated by the perforin pathway, whereas MHC class II-dependent elimination is almost entirely mediated by Fas/FasL interactions (Graubert et al., 1997; Schulz et al., 1995) .
T61652 0-341 Sentence denotes Although cultured CD4 + and CD8 + T-cells can engage both the perforin and the Fas/FasL pathways, in vivo experiments suggest that MHC class I-dependent killing is dominated by the perforin pathway, whereas MHC class II-dependent elimination is almost entirely mediated by Fas/FasL interactions (Graubert et al., 1997; Schulz et al., 1995) .

Epistemic_Statements

Id Subject Object Predicate Lexical cue
T71 0-341 Epistemic_statement denotes Although cultured CD4 + and CD8 + T-cells can engage both the perforin and the Fas/FasL pathways, in vivo experiments suggest that MHC class I-dependent killing is dominated by the perforin pathway, whereas MHC class II-dependent elimination is almost entirely mediated by Fas/FasL interactions (Graubert et al., 1997; Schulz et al., 1995) .