CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 15011-15317 JSONTXT

{"project":"CORD-19-SciBite-sentences","denotations":[{"id":"T151","span":{"begin":0,"end":306},"obj":"http://purl.obolibrary.org/obo/HP0011096"},{"id":"T153","span":{"begin":0,"end":306},"obj":"http://purl.obolibrary.org/obo/GO0030424"},{"id":"T158","span":{"begin":0,"end":306},"obj":"https://id.nlm.nih.gov/mesh/D003711"}],"attributes":[{"id":"A151","pred":"class","subj":"T151","obj":"HPO"},{"id":"A153","pred":"class","subj":"T153","obj":"GOONTOL"},{"id":"A158","pred":"class","subj":"T158","obj":"INDICATION"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T81","span":{"begin":0,"end":306},"obj":"Sentence"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T81","span":{"begin":0,"end":306},"obj":"Sentence"},{"id":"TextSentencer_T81","span":{"begin":0,"end":306},"obj":"Sentence"},{"id":"T18046","span":{"begin":0,"end":306},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}

{"project":"Epistemic_Statements","denotations":[{"id":"T40","span":{"begin":0,"end":306},"obj":"Epistemic_statement"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T59","span":{"begin":279,"end":281},"obj":"Disease"},{"id":"T60","span":{"begin":303,"end":305},"obj":"Disease"}],"attributes":[{"id":"A59","pred":"mondo_id","subj":"T59","obj":"http://purl.obolibrary.org/obo/MONDO_0005301"},{"id":"A60","pred":"mondo_id","subj":"T60","obj":"http://purl.obolibrary.org/obo/MONDO_0005301"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T38","span":{"begin":35,"end":48},"obj":"Phenotype"},{"id":"T39","span":{"begin":128,"end":141},"obj":"Phenotype"},{"id":"T40","span":{"begin":237,"end":250},"obj":"Phenotype"}],"attributes":[{"id":"A38","pred":"hp_id","subj":"T38","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A39","pred":"hp_id","subj":"T39","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A40","pred":"hp_id","subj":"T40","obj":"http://purl.obolibrary.org/obo/HP_0011096"}],"text":"Because the methods used to induce demyelination in these models are extremely disparate, and because the cellular effectors of demyelination are variable across each of these models, it seems that axon failure, rather than mechanism of demyelination, is the critical feature of MS and animal models of MS."}