CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 14736-15010 JSONTXT

{"project":"CORD-19-SciBite-sentences","denotations":[{"id":"T146","span":{"begin":0,"end":274},"obj":"http://purl.obolibrary.org/obo/HP0040078"},{"id":"T150","span":{"begin":0,"end":274},"obj":"http://purl.obolibrary.org/obo/HP0011096"},{"id":"T152","span":{"begin":0,"end":274},"obj":"http://purl.obolibrary.org/obo/GO0030424"},{"id":"T156","span":{"begin":0,"end":274},"obj":"http://purl.obolibrary.org/obo/GO0043526"},{"id":"T157","span":{"begin":0,"end":274},"obj":"https://id.nlm.nih.gov/mesh/D003711"}],"attributes":[{"id":"A146","pred":"class","subj":"T146","obj":"HPO"},{"id":"A150","pred":"class","subj":"T150","obj":"HPO"},{"id":"A152","pred":"class","subj":"T152","obj":"GOONTOL"},{"id":"A156","pred":"class","subj":"T156","obj":"GOONTOL"},{"id":"A157","pred":"class","subj":"T157","obj":"INDICATION"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T79","span":{"begin":0,"end":186},"obj":"Sentence"},{"id":"T80","span":{"begin":187,"end":274},"obj":"Sentence"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T79","span":{"begin":0,"end":186},"obj":"Sentence"},{"id":"TextSentencer_T80","span":{"begin":187,"end":274},"obj":"Sentence"},{"id":"TextSentencer_T79","span":{"begin":0,"end":186},"obj":"Sentence"},{"id":"TextSentencer_T80","span":{"begin":187,"end":274},"obj":"Sentence"},{"id":"T49121","span":{"begin":0,"end":186},"obj":"Sentence"},{"id":"T31539","span":{"begin":187,"end":274},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}

{"project":"Epistemic_Statements","denotations":[{"id":"T38","span":{"begin":0,"end":186},"obj":"Epistemic_statement"},{"id":"T39","span":{"begin":187,"end":274},"obj":"Epistemic_statement"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T58","span":{"begin":221,"end":242},"obj":"Disease"}],"attributes":[{"id":"A58","pred":"mondo_id","subj":"T58","obj":"http://purl.obolibrary.org/obo/MONDO_0002562"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T35","span":{"begin":57,"end":70},"obj":"Phenotype"},{"id":"T36","span":{"begin":171,"end":184},"obj":"Phenotype"},{"id":"T37","span":{"begin":221,"end":234},"obj":"Phenotype"}],"attributes":[{"id":"A35","pred":"hp_id","subj":"T35","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A36","pred":"hp_id","subj":"T36","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A37","pred":"hp_id","subj":"T37","obj":"http://purl.obolibrary.org/obo/HP_0011096"}],"text":"Remyelination as Neuroprotection associated with chronic demyelination induced by cuprizone treatment, with axon diameter being reduced to 60% of normal after 16 weeks of demyelination . Thus, every major animal model of demyelinating disease is associated with axon damage."}