CORD-19:12af471f81b05440047e30b963b3733bd6f33693 / 30556-30905
Annnotations
CORD-19_Custom_license_subset
{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T148","span":{"begin":0,"end":140},"obj":"Sentence"},{"id":"T149","span":{"begin":141,"end":349},"obj":"Sentence"}],"text":"This theory aims to explain interindividual variability to infections by identifying four genetic mechanisms at play in infectious diseases: Mendelian monogenic predisposition to one infection, Mendelian monogenic predisposition to several infections, major gene/resistance to one infection and polygenic predisposition to one infection ( Table 2 )."}
CORD-19-Sentences
{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T148","span":{"begin":0,"end":140},"obj":"Sentence"},{"id":"TextSentencer_T149","span":{"begin":141,"end":349},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"This theory aims to explain interindividual variability to infections by identifying four genetic mechanisms at play in infectious diseases: Mendelian monogenic predisposition to one infection, Mendelian monogenic predisposition to several infections, major gene/resistance to one infection and polygenic predisposition to one infection ( Table 2 )."}
Epistemic_Statements
{"project":"Epistemic_Statements","denotations":[{"id":"T86","span":{"begin":0,"end":349},"obj":"Epistemic_statement"}],"text":"This theory aims to explain interindividual variability to infections by identifying four genetic mechanisms at play in infectious diseases: Mendelian monogenic predisposition to one infection, Mendelian monogenic predisposition to several infections, major gene/resistance to one infection and polygenic predisposition to one infection ( Table 2 )."}
CORD-19-PD-MONDO
{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T79","span":{"begin":59,"end":69},"obj":"Disease"},{"id":"T80","span":{"begin":120,"end":130},"obj":"Disease"},{"id":"T81","span":{"begin":183,"end":192},"obj":"Disease"},{"id":"T82","span":{"begin":240,"end":250},"obj":"Disease"},{"id":"T83","span":{"begin":281,"end":290},"obj":"Disease"},{"id":"T84","span":{"begin":327,"end":336},"obj":"Disease"},{"id":"T16891","span":{"begin":59,"end":69},"obj":"Disease"},{"id":"T6705","span":{"begin":120,"end":130},"obj":"Disease"},{"id":"T3831","span":{"begin":183,"end":192},"obj":"Disease"},{"id":"T15292","span":{"begin":240,"end":250},"obj":"Disease"},{"id":"T31974","span":{"begin":281,"end":290},"obj":"Disease"},{"id":"T60609","span":{"begin":327,"end":336},"obj":"Disease"}],"attributes":[{"id":"A79","pred":"mondo_id","subj":"T79","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A80","pred":"mondo_id","subj":"T80","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A81","pred":"mondo_id","subj":"T81","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A82","pred":"mondo_id","subj":"T82","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A83","pred":"mondo_id","subj":"T83","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A84","pred":"mondo_id","subj":"T84","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A37019","pred":"mondo_id","subj":"T16891","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A85102","pred":"mondo_id","subj":"T6705","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A50646","pred":"mondo_id","subj":"T3831","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A21183","pred":"mondo_id","subj":"T15292","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A64278","pred":"mondo_id","subj":"T31974","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A98396","pred":"mondo_id","subj":"T60609","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"}],"text":"This theory aims to explain interindividual variability to infections by identifying four genetic mechanisms at play in infectious diseases: Mendelian monogenic predisposition to one infection, Mendelian monogenic predisposition to several infections, major gene/resistance to one infection and polygenic predisposition to one infection ( Table 2 )."}