CORD-19:11372d2b62ec98cab9cf3f9be6b7cf967627a14a / 1509-1849 JSONTXT

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T9","span":{"begin":0,"end":340},"obj":"Sentence"}],"text":"In Brief Jiang et al. reveal that CARMA3, a gene located in a host genomic locus that contributes to the host's susceptibility to RNA respiratory virus infection, is a key molecule that controls the balance of proinflammatory and antiviral responses, through positively regulating NF-kB activation but negatively regulating IRF3 activation."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T9","span":{"begin":0,"end":340},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"In Brief Jiang et al. reveal that CARMA3, a gene located in a host genomic locus that contributes to the host's susceptibility to RNA respiratory virus infection, is a key molecule that controls the balance of proinflammatory and antiviral responses, through positively regulating NF-kB activation but negatively regulating IRF3 activation."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T15","span":{"begin":146,"end":161},"obj":"Disease"},{"id":"T16","span":{"begin":152,"end":161},"obj":"Disease"},{"id":"T96936","span":{"begin":146,"end":161},"obj":"Disease"},{"id":"T55352","span":{"begin":152,"end":161},"obj":"Disease"}],"attributes":[{"id":"A15","pred":"mondo_id","subj":"T15","obj":"http://purl.obolibrary.org/obo/MONDO_0005108"},{"id":"A16","pred":"mondo_id","subj":"T16","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"},{"id":"A81078","pred":"mondo_id","subj":"T96936","obj":"http://purl.obolibrary.org/obo/MONDO_0005108"},{"id":"A3292","pred":"mondo_id","subj":"T55352","obj":"http://purl.obolibrary.org/obo/MONDO_0005550"}],"text":"In Brief Jiang et al. reveal that CARMA3, a gene located in a host genomic locus that contributes to the host's susceptibility to RNA respiratory virus infection, is a key molecule that controls the balance of proinflammatory and antiviral responses, through positively regulating NF-kB activation but negatively regulating IRF3 activation."}