CORD-19:02d18b4f484a0420c21595a95b8765125d4a484c / 41558-41730 JSONTXT

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T235","span":{"begin":0,"end":172},"obj":"Sentence"}],"text":"Among the human Hartnup mutations of B 0 AT1, the A69T and R240Q missense mutants were shown in vitro defective for activation of transport function by ACE2 and Collectrin."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T235","span":{"begin":0,"end":172},"obj":"Sentence"},{"id":"TextSentencer_T235","span":{"begin":0,"end":172},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Among the human Hartnup mutations of B 0 AT1, the A69T and R240Q missense mutants were shown in vitro defective for activation of transport function by ACE2 and Collectrin."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T271","span":{"begin":41,"end":44},"obj":"Disease"}],"attributes":[{"id":"A271","pred":"mondo_id","subj":"T271","obj":"http://purl.obolibrary.org/obo/MONDO_0008840"}],"text":"Among the human Hartnup mutations of B 0 AT1, the A69T and R240Q missense mutants were shown in vitro defective for activation of transport function by ACE2 and Collectrin."}