CORD-19:02d18b4f484a0420c21595a95b8765125d4a484c / 28459-28774
Annnotations
CORD-19_Custom_license_subset
{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T167","span":{"begin":0,"end":315},"obj":"Sentence"}],"text":"In humans, ACE2 gene polymorphisms associate with parameters of left ventricular hypertrophy in men (Lieb et al., 2006) , and soluble ACE2 activity is increased in patients with myocardial dysfunction and correlates with disease severity (Epelman et al., 2008) , implicating physiological roles of ACE2 in patients."}
CORD-19-Sentences
{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T167","span":{"begin":0,"end":315},"obj":"Sentence"},{"id":"TextSentencer_T167","span":{"begin":0,"end":315},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"In humans, ACE2 gene polymorphisms associate with parameters of left ventricular hypertrophy in men (Lieb et al., 2006) , and soluble ACE2 activity is increased in patients with myocardial dysfunction and correlates with disease severity (Epelman et al., 2008) , implicating physiological roles of ACE2 in patients."}
Epistemic_Statements
{"project":"Epistemic_Statements","denotations":[{"id":"T60","span":{"begin":0,"end":315},"obj":"Epistemic_statement"}],"text":"In humans, ACE2 gene polymorphisms associate with parameters of left ventricular hypertrophy in men (Lieb et al., 2006) , and soluble ACE2 activity is increased in patients with myocardial dysfunction and correlates with disease severity (Epelman et al., 2008) , implicating physiological roles of ACE2 in patients."}
CORD-19-PD-HP
{"project":"CORD-19-PD-HP","denotations":[{"id":"T38","span":{"begin":64,"end":92},"obj":"Phenotype"}],"attributes":[{"id":"A38","pred":"hp_id","subj":"T38","obj":"http://purl.obolibrary.org/obo/HP_0001712"}],"text":"In humans, ACE2 gene polymorphisms associate with parameters of left ventricular hypertrophy in men (Lieb et al., 2006) , and soluble ACE2 activity is increased in patients with myocardial dysfunction and correlates with disease severity (Epelman et al., 2008) , implicating physiological roles of ACE2 in patients."}