| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T838 |
0-252 |
Sentence |
denotes |
Intracerebral hemorrhage (ICH) represents 10%–15% of all strokes and occurs with increasing frequency as a complication of the thrombolytic treatment of ischemic stroke, which is associated with high mortality and severe disability in survivors [1, 2]. |
| T13 |
0-252 |
Sentence |
denotes |
Intracerebral hemorrhage (ICH) represents 10%–15% of all strokes and occurs with increasing frequency as a complication of the thrombolytic treatment of ischemic stroke, which is associated with high mortality and severe disability in survivors [1, 2]. |
| T839 |
253-472 |
Sentence |
denotes |
Local brain tissue deformation induces primary brain damage after ICH; more importantly, the excitotoxicity and inflammatory responses in the brain tissue induced by ICH result in further brain damage following ICH [3]. |
| T14 |
253-472 |
Sentence |
denotes |
Local brain tissue deformation induces primary brain damage after ICH; more importantly, the excitotoxicity and inflammatory responses in the brain tissue induced by ICH result in further brain damage following ICH [3]. |
| T840 |
473-632 |
Sentence |
denotes |
Both innate and adaptive immunological/inflammatory mechanisms have been clearly shown to participate in the progression of brain injury after ischemic stroke. |
| T15 |
473-632 |
Sentence |
denotes |
Both innate and adaptive immunological/inflammatory mechanisms have been clearly shown to participate in the progression of brain injury after ischemic stroke. |
| T841 |
633-738 |
Sentence |
denotes |
Nevertheless, how the inflammatory responses in ICH are triggered and regulated remains to be elucidated. |
| T16 |
633-738 |
Sentence |
denotes |
Nevertheless, how the inflammatory responses in ICH are triggered and regulated remains to be elucidated. |
