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PMC:1892049 / 7020-7617 JSONTXT 5 Projects

Annnotations TAB TSV DIC JSON TextAE-old TextAE

Id Subject Object Predicate Lexical cue
T6925 0-5 VBN denotes Given
T6927 6-9 PRP$ denotes our
T6928 10-18 NN denotes interest
T6929 19-21 IN denotes in
T6930 22-27 JJ denotes human
T6932 28-40 JJ denotes neurological
T6931 41-48 NN denotes disease
T6933 49-51 PRP denotes we
T6926 52-58 VBD denotes sought
T6934 59-61 TO denotes to
T6935 62-70 VB denotes identify
T6936 71-74 DT denotes any
T6938 75-82 JJ denotes cognate
T6939 83-88 JJ denotes human
T6937 89-98 NNS denotes disorders
T6940 99-104 WRB denotes where
T6942 105-112 NN denotes linkage
T6943 113-116 VBD denotes had
T6944 117-121 VBN denotes been
T6941 122-133 VBN denotes established
T6945 134-136 IN denotes to
T6946 137-140 DT denotes the
T6948 141-149 JJ denotes syntenic
T6947 150-156 NN denotes region
T6949 157-159 IN denotes of
T6950 160-163 DT denotes the
T6952 164-169 JJ denotes human
T6951 170-176 NN denotes genome
T6953 176-178 , denotes ,
T6954 178-181 CC denotes but
T6955 182-187 WRB denotes where
T6957 188-190 DT denotes no
T6959 191-197 JJ denotes causal
T6958 198-206 NN denotes mutation
T6960 207-210 VBD denotes had
T6961 211-215 VBN denotes been
T6956 216-226 VBN denotes identified
T6962 226-227 . denotes .
T6963 227-316 sentence denotes SCA15, an adult-onset autosomal dominant progressive ataxia is linked to this locus [5].
T6964 228-233 NN denotes SCA15
T6966 233-235 , denotes ,
T6967 235-237 DT denotes an
T6969 238-243 JJ denotes adult
T6971 243-244 HYPH denotes -
T6970 244-249 NN denotes onset
T6972 250-259 JJ denotes autosomal
T6973 260-268 JJ denotes dominant
T6974 269-280 JJ denotes progressive
T6968 281-287 NN denotes ataxia
T6975 288-290 VBZ denotes is
T6965 291-297 VBN denotes linked
T6976 298-300 IN denotes to
T6977 301-305 DT denotes this
T6978 306-311 NN denotes locus
T6979 312-313 -LRB- denotes [
T6980 313-314 CD denotes 5
T6981 314-315 -RRB- denotes ]
T6982 315-316 . denotes .
T6983 316-597 sentence denotes Although missense mutation of ITPR1 had previously been ruled out [2] and the mode of inheritance was inconsistent with that seen in the Itpr1Δ18 and Itpr1opt mice, the phenotypic presence of ataxia in the mice led us to reexamine this candidate gene as a possible cause of SCA15.
T6984 317-325 IN denotes Although
T6986 326-334 NN denotes missense
T6987 335-343 NN denotes mutation
T6988 344-346 IN denotes of
T6989 347-352 NN denotes ITPR1
T6990 353-356 VBD denotes had
T6991 357-367 RB denotes previously
T6992 368-372 VBN denotes been
T6985 373-378 VBN denotes ruled
T6994 379-382 RP denotes out
T6995 383-384 -LRB- denotes [
T6996 384-385 CD denotes 2
T6997 385-386 -RRB- denotes ]
T6998 387-390 CC denotes and
T6999 391-394 DT denotes the
T7000 395-399 NN denotes mode
T7002 400-402 IN denotes of
T7003 403-414 NN denotes inheritance
T7001 415-418 VBD denotes was
T7004 419-431 JJ denotes inconsistent
T7005 432-436 IN denotes with
T7006 437-441 DT denotes that
T7007 442-446 VBN denotes seen
T7008 447-449 IN denotes in
T7009 450-453 DT denotes the
T7011 454-462 NN denotes Itpr1Δ18
T7012 463-466 CC denotes and
T7013 467-475 NN denotes Itpr1opt
T7010 476-480 NNS denotes mice
T7014 480-482 , denotes ,
T7015 482-485 DT denotes the
T7017 486-496 JJ denotes phenotypic
T7016 497-505 NN denotes presence
T7018 506-508 IN denotes of
T7019 509-515 NN denotes ataxia
T7020 516-518 IN denotes in
T7021 519-522 DT denotes the
T7022 523-527 NNS denotes mice
T6993 528-531 VBD denotes led
T7023 532-534 PRP denotes us
T7024 535-537 TO denotes to
T7025 538-547 VB denotes reexamine
T7026 548-552 DT denotes this
T7028 553-562 NN denotes candidate
T7027 563-567 NN denotes gene
T7029 568-570 IN denotes as
T7030 571-572 DT denotes a
T7032 573-581 JJ denotes possible
T7031 582-587 NN denotes cause
T7033 588-590 IN denotes of
T7034 591-596 NN denotes SCA15
T7035 596-597 . denotes .
R1001 T7027 T7025 dobj gene,reexamine
R1002 T7028 T7027 compound candidate,gene
R1004 T7029 T7025 prep as,reexamine
R1005 T7030 T7031 det a,cause
R1006 T7031 T7029 pobj cause,as
R1008 T7032 T7031 amod possible,cause
R1009 T7033 T7031 prep of,cause
R1011 T7034 T7033 pobj SCA15,of
R1012 T7035 T6993 punct .,led
R863 T6925 T6926 prep Given,sought
R864 T6927 T6928 poss our,interest
R865 T6928 T6925 pobj interest,Given
R866 T6929 T6928 prep in,interest
R867 T6930 T6931 amod human,disease
R869 T6931 T6929 pobj disease,in
R870 T6932 T6931 amod neurological,disease
R873 T6933 T6926 nsubj we,sought
R874 T6934 T6935 aux to,identify
R877 T6935 T6926 xcomp identify,sought
R879 T6936 T6937 det any,disorders
R880 T6937 T6935 dobj disorders,identify
R882 T6938 T6937 amod cognate,disorders
R883 T6939 T6937 amod human,disorders
R885 T6940 T6941 advmod where,established
R887 T6941 T6937 advcl established,disorders
R888 T6942 T6941 nsubjpass linkage,established
R889 T6943 T6941 aux had,established
R891 T6944 T6941 auxpass been,established
R892 T6945 T6941 prep to,established
R894 T6946 T6947 det the,region
R895 T6947 T6945 pobj region,to
R896 T6948 T6947 amod syntenic,region
R898 T6949 T6947 prep of,region
R899 T6950 T6951 det the,genome
R900 T6951 T6949 pobj genome,of
R902 T6952 T6951 amod human,genome
R903 T6953 T6941 punct ", ",established
R904 T6954 T6941 cc but,established
R906 T6955 T6956 advmod where,identified
R907 T6956 T6941 conj identified,established
R909 T6957 T6958 det no,mutation
R910 T6958 T6956 nsubjpass mutation,identified
R911 T6959 T6958 amod causal,mutation
R913 T6960 T6956 aux had,identified
R914 T6961 T6956 auxpass been,identified
R915 T6962 T6926 punct .,sought
R917 T6964 T6965 nsubjpass SCA15,linked
R918 T6966 T6964 punct ", ",SCA15
R919 T6967 T6968 det an,ataxia
R920 T6968 T6964 appos ataxia,SCA15
R921 T6969 T6970 amod adult,onset
R922 T6970 T6968 nmod onset,ataxia
R924 T6971 T6970 punct -,onset
R925 T6972 T6973 amod autosomal,dominant
R926 T6973 T6968 amod dominant,ataxia
R928 T6974 T6968 amod progressive,ataxia
R929 T6975 T6965 auxpass is,linked
R930 T6976 T6965 prep to,linked
R932 T6977 T6978 det this,locus
R933 T6978 T6976 pobj locus,to
R935 T6979 T6980 punct [,5
R936 T6980 T6965 parataxis 5,linked
R938 T6981 T6980 punct ],5
R939 T6982 T6965 punct .,linked
R941 T6984 T6985 mark Although,ruled
R942 T6985 T6993 advcl ruled,led
R943 T6986 T6987 compound missense,mutation
R945 T6987 T6985 nsubjpass mutation,ruled
R946 T6988 T6987 prep of,mutation
R947 T6989 T6988 pobj ITPR1,of
R949 T6990 T6985 aux had,ruled
R950 T6991 T6985 advmod previously,ruled
R951 T6992 T6985 auxpass been,ruled
R953 T6994 T6985 prt out,ruled
R954 T6995 T6996 punct [,2
R956 T6996 T6985 parataxis 2,ruled
R957 T6997 T6996 punct ],2
R958 T6998 T6985 cc and,ruled
R960 T6999 T7000 det the,mode
R961 T7000 T7001 nsubj mode,was
R964 T7001 T6985 conj was,ruled
R966 T7002 T7000 prep of,mode
R967 T7003 T7002 pobj inheritance,of
R968 T7004 T7001 acomp inconsistent,was
R969 T7005 T7004 prep with,inconsistent
R970 T7006 T7005 pobj that,with
R971 T7007 T7006 acl seen,that
R973 T7008 T7007 prep in,seen
R974 T7009 T7010 det the,mice
R975 T7010 T7008 pobj mice,in
R977 T7011 T7010 nmod Itpr1Δ18,mice
R978 T7012 T7011 cc and,Itpr1Δ18
R979 T7013 T7011 conj Itpr1opt,Itpr1Δ18
R981 T7014 T6993 punct ", ",led
R982 T7015 T7016 det the,presence
R983 T7016 T6993 nsubj presence,led
R985 T7017 T7016 amod phenotypic,presence
R988 T7018 T7016 prep of,presence
R989 T7019 T7018 pobj ataxia,of
R991 T7020 T7016 prep in,presence
R992 T7021 T7022 det the,mice
R994 T7022 T7020 pobj mice,in
R995 T7023 T6993 dobj us,led
R997 T7024 T7025 aux to,reexamine
R998 T7025 T6993 xcomp reexamine,led
R999 T7026 T7027 det this,gene