PubMed:23390526 / 1071-1309 JSONTXT

{"project":"Inflammaging","denotations":[{"id":"T9","span":{"begin":0,"end":238},"obj":"Sentence"},{"id":"T9","span":{"begin":0,"end":238},"obj":"Sentence"}],"text":"One of those QTL was significant at the genome-wide level and largely explained both survival and viral replication in fin explants in the different families of the design (up to 65% and 49% of phenotypic variance explained respectively)."}

{"project":"PubMed_ArguminSci","denotations":[{"id":"T8","span":{"begin":0,"end":238},"obj":"DRI_Background"}],"text":"One of those QTL was significant at the genome-wide level and largely explained both survival and viral replication in fin explants in the different families of the design (up to 65% and 49% of phenotypic variance explained respectively)."}

{"project":"Goldhamster2_Cellosaurus","denotations":[{"id":"T16","span":{"begin":0,"end":3},"obj":"CVCL_Z424|Spontaneously_immortalized_cell_line|Ostrinia nubilalis"},{"id":"T17","span":{"begin":104,"end":115},"obj":"CVCL_XY58|Transformed_cell_line|Mus musculus"},{"id":"T18","span":{"begin":119,"end":122},"obj":"CVCL_W752|Cancer_cell_line|Homo sapiens"},{"id":"T19","span":{"begin":187,"end":189},"obj":"CVCL_UU95|Spontaneously_immortalized_cell_line|Cricetulus griseus"}],"text":"One of those QTL was significant at the genome-wide level and largely explained both survival and viral replication in fin explants in the different families of the design (up to 65% and 49% of phenotypic variance explained respectively)."}

{"project":"GoldHamster","denotations":[{"id":"T44","span":{"begin":13,"end":16},"obj":"SO:0000771"},{"id":"T45","span":{"begin":40,"end":46},"obj":"SO:0001026"},{"id":"T46","span":{"begin":98,"end":115},"obj":"GO:0019058"},{"id":"T47","span":{"begin":98,"end":115},"obj":"GO:0019079"},{"id":"T48","span":{"begin":98,"end":115},"obj":"GO:0008166"},{"id":"T50","span":{"begin":119,"end":122},"obj":"CVCL_W752"},{"id":"T49","span":{"begin":119,"end":122},"obj":"UBERON:0008897"}],"text":"One of those QTL was significant at the genome-wide level and largely explained both survival and viral replication in fin explants in the different families of the design (up to 65% and 49% of phenotypic variance explained respectively)."}