PubMed:22891926 / 65-331 JSONTXT

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    PubMed_Structured_Abstracts

    {"project":"PubMed_Structured_Abstracts","denotations":[{"id":"T1","span":{"begin":0,"end":266},"obj":"BACKGROUND"}],"text":"Diabetic tendinopathy is characterized by increased stiffness, thickness, and excess calcification of affected tendons. We investigated the hypothesis that proteoglycans (PGs), as key regulators of tendon structure and calcification, are altered in diabetic tendons."}

    Goldhamster2_Cellosaurus

    {"project":"Goldhamster2_Cellosaurus","denotations":[{"id":"T1","span":{"begin":120,"end":122},"obj":"CVCL_5M23|Cancer_cell_line|Mesocricetus auratus"}],"text":"Diabetic tendinopathy is characterized by increased stiffness, thickness, and excess calcification of affected tendons. We investigated the hypothesis that proteoglycans (PGs), as key regulators of tendon structure and calcification, are altered in diabetic tendons."}

    GoldHamster

    {"project":"GoldHamster","denotations":[{"id":"T7","span":{"begin":9,"end":21},"obj":"D052256"},{"id":"T8","span":{"begin":9,"end":21},"obj":"D052256"},{"id":"T9","span":{"begin":156,"end":169},"obj":"CHEBI:37396"},{"id":"T10","span":{"begin":156,"end":169},"obj":"D011509"},{"id":"T11","span":{"begin":156,"end":169},"obj":"D011509"},{"id":"T12","span":{"begin":171,"end":174},"obj":"C535773"},{"id":"T13","span":{"begin":198,"end":204},"obj":"UBERON:0000043"}],"text":"Diabetic tendinopathy is characterized by increased stiffness, thickness, and excess calcification of affected tendons. We investigated the hypothesis that proteoglycans (PGs), as key regulators of tendon structure and calcification, are altered in diabetic tendons."}

    Allie

    {"project":"Allie","denotations":[{"id":"SS1_22891926_3_0","span":{"begin":156,"end":169},"obj":"expanded"},{"id":"SS2_22891926_3_0","span":{"begin":171,"end":174},"obj":"abbr"}],"relations":[{"id":"AE1_22891926_3_0","pred":"abbreviatedTo","subj":"SS1_22891926_3_0","obj":"SS2_22891926_3_0"}],"text":"Diabetic tendinopathy is characterized by increased stiffness, thickness, and excess calcification of affected tendons. We investigated the hypothesis that proteoglycans (PGs), as key regulators of tendon structure and calcification, are altered in diabetic tendons."}