PMC:88885 / 2879-3051 JSONTXT

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{"project":"craft-ca-core-dev","denotations":[{"id":"T544","span":{"begin":15,"end":24},"obj":"GO:0005764"},{"id":"T545","span":{"begin":60,"end":82},"obj":"NCBITaxon:6239"},{"id":"T546","span":{"begin":141,"end":147},"obj":"PR:000010252"},{"id":"T547","span":{"begin":148,"end":158},"obj":"NCBITaxon:6239"},{"id":"T548","span":{"begin":159,"end":168},"obj":"SO:0000853"}],"text":"d by defective lysosomal hydrolases [11,12]. Recent work in Caenorhabditis elegans supports this hypothesis. Loss of function mutants of the MCOLN1 C. elegans homologue, cu"}

{"project":"2_test","denotations":[{"id":"11897010-9323557-10449844","span":{"begin":37,"end":39},"obj":"9323557"},{"id":"11897010-9600972-10449845","span":{"begin":40,"end":42},"obj":"9600972"}],"text":"d by defective lysosomal hydrolases [11,12]. Recent work in Caenorhabditis elegans supports this hypothesis. Loss of function mutants of the MCOLN1 C. elegans homologue, cu"}

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