Among patients diagnosed with SARS-CoV-2, it has been reported that renal dysfunction is characterized by high levels of blood urea nitrogen, creatinine, uric acid, and D-dimer, associated with proteinuria and hematuria (90, 117–119). Recent studies have reported an incidence between 3-9% of acute kidney injury in COVID-19 patients, demonstrating renal abnormalities (94, 96, 111, 120). Cardiovascular complications are also associated with COVID-19 infection, including myocardial injury, myocarditis, acute myocardial infarction, heart failure, dysrhythmias, and venous thromboembolic events, being significant contributors to the mortality associated with this disease (121, 122).